Risk Factors and Clinical Outcomes of Glucose-6-Phosphate Dehydrogenase Deficiency in Neonates: A Case-Control Study

新生儿葡萄糖-6-磷酸脱氢酶缺乏症的危险因素和临床结局:一项病例对照研究

阅读:1

Abstract

Background Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a common inherited enzymatic disorder and a major risk factor for neonatal hyperbilirubinemia. This study aims to identify baseline risk factors and evaluate clinical outcomes associated with G6PD deficiency in neonates. Methods A case-control study was conducted, including 150 neonates, comprising 50 G6PD-deficient cases and 100 controls. Demographic characteristics, clinical variables, and laboratory parameters were compared between groups. Categorical variables were analyzed using chi-square (χ²) tests, and continuous variables were analyzed using the Mann-Whitney U test. A p-value <0.05 was considered statistically significant. Results Significant associations were observed between G6PD deficiency and male sex, ethnicity, family history of G6PD deficiency, prolonged hospital stay, phototherapy requirement, and presence of jaundice (p <0.001). G6PD-deficient neonates had significantly higher total bilirubin levels and lower G6PD enzyme levels compared with controls. No significant associations were found with gestational age, feeding type, mode of delivery, or maternal and fetal complications. Conclusions G6PD deficiency was significantly associated with key risk factors like male gender, ethnicity, family history of G6PD, and clinical outcomes, including neonatal hyperbilirubinemia, jaundice, prolonged hospitalization, and increased phototherapy requirement.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。