Establishment and validation of a novel risk model based on PANoptosis-related genes to predict prognosis in head and neck squamous cell carcinoma

建立并验证基于PANoptosis相关基因的新型风险模型,用于预测头颈部鳞状细胞癌的预后

阅读:1

Abstract

Head and neck squamous cell carcinoma (HNSC) is a common cancer worldwide with poor prognosis. Current treatment methods have limited effect on improving the prognosis of patients with HNSC. Differentially expressed PANoptosis-related genes in HNSC were identified from the TCGA using limma and WGCNA. A prognostic model was established using univariate and multivariate Cox regression analyses and machine learning, and its performance was evaluated using Kaplan-Meier and receiver operating characteristic curves. SNP data was analyzed using maftools package. Immune analysis was performed using IOBR package and TIDE website. The scRNA data was analyzed using Seurat and cellchat package. The expression of hub genes was validated in vitro. The prognostic model comprising 5 hub PANoptosis-related genes (AIFM1, AKT3, CDKN2A, EGFR, IL1RAP) accurately predicted patient outcomes, with the high-risk group exhibiting poorer survival. mRNA expression levels of all 5 hub genes were elevated in the tumor cells, but only AIFM1, AKT3 and IL1RAP's protein expression were higher in tumor tissues. Additionally, high expression of AIFM1, AKT3, EGFR, IL1RAP and low expression of CDKN2A indicated poor prognosis of HNSC patients. The decreasing levels of CD4 T cells, CD8 T cells and M1 macrophages were observed in high-risk groups. There was a significant difference of 5-fluorouracil in low and high-risk groups. scRNA analysis exhibited that TNF pathway was important in the interaction between macrophages and T cells. We identified 5 hub genes and constructed a great risk model for the prognosis of HNSC. The immune cells may influence the HNSC malignant through TNF signal pathway.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。