β-Lapachone encapsulated into stealth liposomes: inhibition of biofilm and cell wall thickness of MRSA

β-拉帕酮包裹于隐形脂质体中:抑制耐甲氧西林金黄色葡萄球菌(MRSA)的生物膜形成和细胞壁增厚

阅读:1

Abstract

Methicillin-resistant Staphylococcus aureus (MRSA) is a biofilm-producing pathogen that causes infections worldwide and is considered a global public health problem. In this scenario, new therapeutic strategies to eliminate this pathogen and eradicate the biofilm are essential. Thus, this study aimed to evaluate the effects of β-lapachone (β-lap) encapsulated into stealth liposomes (β-lap-SL) on biofilm and cell wall thickness of MRSA, as well as their hemotoxicity. β-lap-SL were prepared using the lipid film hydration method and physicochemical characteristics were determined. Antibiofilm activity was measured through the crystal violet staining method, the changes in cell wall thickness of MRSA were verified by transmission electron microscopy, and the biocompatibility of the formulation was assessed by hemolytic assay. β-lap-SL was produced and exhibited particle size of 84.2 ± 8.9 nm, polydispersion index of 0.28, zeta potential of + 0.31 ± 0.06 mV, encapsulation efficiency of 98.7 ± 0.6% and pH of 7.5. The antibiofilm activity of β-lap-SL showed values ​​between 4 and 32 µg/mL, and β-lap-SL inhibits the cell wall thickness (55%). β-lap at 64 µg/mL exhibited high toxicity in erythrocytes (~ 45%) compared to β-lap-SL (< 5%) at the same concentration. Based on the results of this study, β-lap-SL appears as a therapeutic agent, and it is a promising option for controlling MRSA-induced infections. SUPPLEMENTARY INFORMATION: The online version contains supplementary material available at 10.1007/s42770-025-01701-1.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。