Assessment of Thiol/Disulfide Homeostasis and Its Association With Clinical Manifestations in Behcet's Disease

评估硫醇/二硫键稳态及其与白塞氏病临床表现的关系

阅读:1

Abstract

Behcet's disease (BD) is a multisystemic inflammatory vasculitis characterized by recurrent episodes of exacerbation and remission, with unpredictable duration and system involvement in each episode. Although previous studies have shown the role of oxidative stress in the pathogenesis of BD, the relationship between the clinical features of Behcet's disease and oxidative stress has not been fully studied to date. This study aims to assess the parameters of dynamic thiol disulfide homeostasis (DTDH) and their relationship with different clinical characteristics of disease in BD patients. Fifty patients with BD and 54 age and gender-matched healthy controls were included in the study. Disease activity was measured by the BD Current Activity Form. The mean total thiol (TT) levels were higher in BD patients compared to controls (p=0.04). The native thiol (NT) levels were lower in patients with BD compared to healthy individuals (p=0.014). Disulfide levels, disulfide/NT, and disulfide/TT levels were similar in both groups (all, p>0.05). No significant differences in DTDH parameters were observed based on the presence or absence of erythema nodosum, papulopustular lesions, venous thrombosis, arthritis/arthralgia, neurological involvement, pathergy test positivity, or HLA-B51 status (all, p>0.05). We found lower NT levels in patients with genital ulcers or eye involvement compared to those without these manifestations (p=0.036 and p=0.003, respectively). We observed similar DTDH parameters in patients with and without active disease, and in patients with and without current medical therapy (all, p>0.05). In addition, we found a negative relationship between disease duration and TT and NT levels (all, p<0.05). In conclusion, we demonstrated an imbalance of DTDH in the patients with BD, supporting the hypothesis that oxidative stress may contribute to the etiopathogenesis and progression of the disease.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。