Impact of stress hyperglycemia mediating tissue-level collaterals on futile recanalization in large vessel occlusion patients

应激性高血糖介导的组织水平侧支循环对大血管闭塞患者无效再通的影响

阅读:1

Abstract

BACKGROUND: The stress hyperglycemia ratio (SHR) is associated with unfavorable functional outcomes in patients with large vessel occlusion. The potential effect of SHR on tissue-level collaterals (TLC) and futile recanalization is not clear. METHODS: This is a multicenter retrospective cohort study of patients with consecutive acute ischemic stroke due to large vessel occlusion (AIS-LVO) receiving endovascular treatment (EVT). The included patients had baseline glucose/HbA1c measurements and underwent cerebral perfusion imaging. TLC were measured using the hypoperfusion intensity ratio (HIR) [the volume ratio of brain tissue with (T (max) > 10 s/T (max) > 6 s)]. SHR was calculated as blood glucose (mmol/L)/[1.59 × HbA1C (%) - 2.59]. Using multivariable regression and mediation analyses, we determined the association among SHR, the TLC status, and futile recanalization (90-day modified Rankin Scale scores 3-6 despite successful recanalization). RESULTS: A total of 246 patients met the inclusion criteria. Patients in the highest tertile of SHR were significantly more likely to suffer futile recanalization compared with those in the lowest tertile [adjusted OR (aOR) = 3.56, 95%CI = 1.73-7.30, p < 0.001]. The TLC (aOR = 3.38, 95%CI = 1.23-9.27, p = 0.018) was worse in patients with elevated SHR and also acted as an independent predictor of futile recanalization (aOR = 2.31, 95%CI = 1.32-4.05, p = 0.003). Mediation analyses showed that the increased SHR was associated with worse TLC, accounting for 9.7% (95%CI = 1.9%-28.0%) of the harmful effect on futile recanalization. Mediation analyses also indicated a partial mediation effect of the baseline larger ischemic core (effect value = 13.5%, 95%CI = 3.1%-32.0%). CONCLUSION: An increased SHR is correlated with unfavorable TLC and is associated with futile recanalization after EVT. Future prospective studies should independently validate our findings.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。