Aortic pressure-velocity loop: a potential tool for assessing risk associated with midsystolic forward compression waves in patients with chronic atherosclerotic coronary artery disease

主动脉压力-速度环:一种评估慢性动脉粥样硬化性冠状动脉疾病患者收缩中期前向压缩波相关风险的潜在工具

阅读:1

Abstract

STUDY OBJECTIVE: To evaluate whether impaired ventriculo-aretrial interaction characterizes chronic ischemic heart disease (CIHD) caused by stable chronic obstructive coronary artery disease (CAD), using wave intensity and aortic pressure-velocity (PU) loop analyses. DESIGN: Retrospective observational study. SETTING: Single-center invasive hemodynamic assessment using high-fidelity multisensor catheters. PARTICIPANTS: Thirty-one patients with CIHD, including 21 with chronic atherosclerotic CAD and 10 with non-CAD CIHD. INTERVENTIONS: None. MAIN OUTCOME MEASURES: Wave intensity-derived indices of ventriculo-arterial interaction, including the initial PU-loop slope (S1), second ascending slope (S2), backward compression wave (BCW), midsystolic forward compression wave (m-FCW), and their relative magnitudes. Discriminatory ability for CAD was assessed using receiver operating characteristic (ROC) curve analysis. RESULTS: In CAD patients, midsystolic flow was supported by a previously unreported m-FCW that emerged in response to an early-onset BCW during ventricular acceleration, reflected by higher S2 values. In contrast, midsystolic flow in non-CAD CIHD patients was primarily maintained by blood inertia and lower S1 values. S2 strongly correlated with the relative magnitude of m-FCW (r = -0.936, P < 0.001). The relative magnitude of m-FCW showed greater discriminatory ability for CAD than BCW (AUC 0.976 vs. 0.771). The S2:S1 ratio correlated closely with the relative magnitude of m-FCW and demonstrated the highest AUC (0.986). CONCLUSIONS: Chronic atherosclerotic CAD is characterized by impaired ventriculo-aortic interaction, with midsystolic flow dependent on compensatory m-FCW rather than blood inertia. Aortic PU-loop parameters, particularly the S2:S1 ratio, may provide a potential diagnostic marker for distinguishing chronic atherosclerotic CAD from other forms of CIHD.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。