Estimated glucose disposal rate and cardiovascular disease risk: a meta-analysis of cohort studies

估计葡萄糖处置率与心血管疾病风险:队列研究的荟萃分析

阅读:1

Abstract

BACKGROUND: Insulin resistance (IR) is a key cardiovascular disease (CVD) risk factor. The estimated glucose disposal rate (eGDR) is a reliable IR marker linked to CVD risk. This study is the first extensive meta-analysis of this correlation in a general population free from baseline CVD. METHODS: We searched electronic databases such as PubMed, Web of Science and Embase for cohort studies reporting eGDR and CVD risk. Studies included adults without baseline CVD, measured eGDR at baseline, and reported hazard ratio (HR) [95% confidence interval (CI)]. The combined HR and its 95% CI were determined through the application of random or fixed effects models. Meta-regression with robust error was utilized to depict the nonlinear dose-response relationship. RESULTS: Twelve cohort studies with 547,287 subjects were included, with follow-up durations ranging from 5.6 to 14.1 years. Participants with the highest eGDR category had a lower risk of CVD (HR: 0.58, 95% CI 0.53-0.63), stroke (HR: 0.62, 95% CI: 0.56-0.69), and coronary heart disease (HR: 0.46, 95% CI: 0.25-0.83) compared with the lowest eGDR category. This aligns with the meta-analysis results, where eGDR as a continuous variable had HRs of 0.88 (95% CI: 0.85-0.91) for CVD, 0.84 (95% CI: 0.76-0.93) for stroke, and 0.85 (95% CI: 0.83-0.87) for coronary heart disease. Subgroup analyses revealed that sex, sample size, follow-up duration, and prediabetes/diabetes status did not significantly affect the results. Dose-response analysis indicated that there was a linear negative association of the eGDR with the risk of CVD (P(nonlinear)=0.120) or stroke (P(nonlinear)=0.084). CONCLUSIONS: The higher eGDR is associated with lower risk of CVD, stroke, and coronary heart disease in individuals without baseline CVD. However, the observational design and high heterogeneity across studies prevent causal inference.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。