The protective effect of biologic and targeted-synthetic therapies on developing multisystem inflammatory syndrome in children

生物制剂和靶向合成疗法对儿童多系统炎症综合征发展的保护作用

阅读:1

Abstract

BACKGROUND: Multisystem Inflammatory Syndrome in Children (MIS-C) is a severe, life threatening, complication that arises weeks after acute Coronavirus disease 2019 (COVID-19) infection, often presenting with fever and diverse systemic symptoms. Limited data exists on the effectiveness of biologic and targeted-synthetic therapies in preventing MIS-C development. Therefore, our aim was to investigate whether biologic and targeted-synthetic therapies can prevent the occurrence of MIS-C. METHODS: We assessed the Clalit Health Services database, the largest health care organization in Israel, data from 793,909 children aged 0-18 years who tested positive for COVID-19 were analyzed. The diagnosis of MIS-C was adjudicated using the case definition used by the Centers for Disease Control and Prevention (CDC) or by the World Health Organization (WHO). Patients receiving biologic and targeted-synthetic therapies were compared to a control group. RESULTS: Among 793,909 cases, 573 children received biologic and targeted-synthetic therapies, and 143 cases of MIS-C were identified. Notably, none of the individuals treated with biologic and targeted-synthetic therapies developed MIS-C. CONCLUSION: Our study highlights our hypothesis on the efficacy of biological treatments in preventing MIS-C. Although statistical significance was not achieved due to the absence of MIS-C cases in patients receiving biologic and targeted-synthetic therapies, our study shows a possible association between biological therapies and reduced risk of MIS-C following COVID-19 infection in children. Further research, including prospective studies with larger cohorts, is warranted to confirm these findings and elucidate underlying mechanisms.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。