Placental miRNA profiling in assisted reproductive technology (ART) pregnancies

辅助生殖技术(ART)妊娠中胎盘miRNA谱分析

阅读:1

Abstract

PURPOSE: Assisted reproductive technology (ART) procedures are associated with placental dysfunction involving dysregulation of several molecular factors and pathways. MicroRNAs (miRNAs) are crucial epigenetic regulators involved in placental development and function. The objective was to identify differential miRNAs in the placentae of women conceived by ART procedures and explore their role by pathway analysis. METHODS: Fifty pregnant women who underwent ART procedure and 50 pregnant women with natural pregnancy (Non-ART group) were included in the study. Qiagen miRCURY LNA PCR Array was used to identify differential miRNAs from placental samples using qRT-PCR. Pathway enrichment analysis was performed for differentially expressed miRNAs using Enrichr integrated within ShinyGO v0.80. RESULTS: Placental expression of 11 hsa-miRNAs (from 28 tested hsa-miRNAs) differed (5 upregulated and 6 downregulated) in the ART group. Pathway enrichment analysis revealed that upregulated miRNAs are associated with pathways such as vasculature development, and downregulated miRNAs were associated with xenobiotic stimulus, stress-activated MAPK cascade, circadian rhythm, insulin stimulus and secretion, cellular response to oxygen levels and fibroblast proliferation. Validation of miRNAs on larger sample size demonstrated that hsa-miR-30c-5p and hsa-miR-140a-5p were downregulated in the ART group. miRNA-mRNA target network analysis revealed that these miRNAs target 58 genes associated with regulation of VEGFA receptor signaling pathway, glomerulus vasculature development, long chain fatty-acyl-CoA biosynthesis, and apoptosis. CONCLUSION: ART procedures are associated with altered placental miRNA expression, possibly contributing to disturbed placental angiogenesis. This study identified two essential miRNAs (hsa-miR-30c-5p and hsa-miR-140a-5p) associated with ART procedures that regulate placental and fetal growth and development.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。