Comprehensive Numerical and Experimental Investigations on Endolaryngeal Mobilization

对喉内松动的综合数值和实验研究

阅读:1

Abstract

OBJECTIVE: To evaluate how subperichondrial mobilization affects implant-tissue mechanics, glottal configuration, and vibratory behavior in computational simulations of medialization laryngoplasty (ML). METHODS: We used a finite-discrete element method (FDEM) framework to simulate subperichondrial tissue-cartilage separation and implant insertion in laryngeal models reconstructed from high-resolution magnetic resonance imaging. Four dissection conditions were evaluated, ranging from no mobilization to increasing dissection length distal to the thyroplasty window. Outcome measures included change in glottal area, vocal fold medial displacement, finite element fracturing along the tissue-cartilage interface, and estimated vibratory frequency. RESULTS: Increasing dissection length produced increased medial displacement, progressive reductions in glottal area, fewer secondary extensions of the dissection plane during implantation, and higher estimated vibratory frequencies (range: 100.4-120.8 Hz). These findings indicate that subperichondrial tissue mobilization alters implant-induced force transmission and modifies boundary conditions relevant to vibration. CONCLUSION: In this first application of FDEM to simulate laryngeal biomechanics in ML, subperichondrial dissection length demonstrated direct effects on model predictions relevant to implant sizing, placement strategy, and anticipated phonatory outcomes. Incorporating tissue mobilization into computational frameworks as a mechanically meaningful variable improves physiological realism and supports future development of subject-specific surgical planning tools. LEVEL OF EVIDENCE: Level V. This study provides preclinical computational evidence using FDEM, supporting and extending clinical observations in ML.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。