Eosinophilic cytokines and small airway dysfunction in women with COPD: correlation with exposure to biomass smoke versus tobacco smoking

慢性阻塞性肺疾病女性患者嗜酸性粒细胞因子和小气道功能障碍:与生物质燃料烟雾暴露和烟草吸烟的相关性

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Abstract

BACKGROUND: Chronic obstructive pulmonary disease (COPD) is a chronic inflammatory disease characterised by progressive airflow obstruction. Tobacco smoking is the leading cause of COPD (COPD-TS); however, chronic exposure to biomass smoke (COPD-BS) is the second risk factor. COPD-TS and COPD-BS are now considered different phenotypes, with COPD-BS having small airway dysfunction (SAD). OBJECTIVES: Our primary aim was to investigate the correlation between SAD and the serum levels of interleukins implicated in the maturation and recruitment of eosinophils in women with COPD-BS and COPD-TS. METHODS: This cross-sectional study included 25 women with COPD-BS and 25 women with COPD-TS, and the relationship with SAD was assessed by impulse oscillometry (IOS), alongside measurements of interleukins (IL-1ra, IL-2, IL-4, IL-8, IL-9, IL-13, and IL-17) and eotaxin using the multiplex test (Bio-Plex), in addition to eosinophils total blood. RESULTS: Blood eosinophil count, cytokines, and eotaxin were higher in COPD-BS relative to COPD-TS. IOS measurements were higher in COPD-BS than in COPD-TS. R5-R20 positively correlated with the concentrations of all measured cytokines noted if COPD groups were analysed separately (P < 0.01). CONCLUSION: Eosinophil counts and cytokines in COPD-BS positively correlate with small airway resistance as measured by IOS in R5-R20.

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