Rekindling the role of plasma Vitamin B(12) levels in individuals with and without oral potentially malignant disorders (OPMDs): An observational study

重新审视血浆维生素B12水平在患有和未患有口腔潜在恶性疾病(OPMDs)的个体中的作用:一项观察性研究

阅读:3

Abstract

BACKGROUND: Vitamin B(12) is a water-soluble vitamin involved in the metabolism of every cell of the human body especially affecting DNA synthesis, fatty acid and amino acid metabolism. Deficiency of this vitamin is usually associated with megaloblastic anaemia, neuropathies or pernicious anaemia. The deficiency most often goes unrecognized because the clinical manifestations are highly variable, often subtle and non-specific. The study of plasma Vitamin B(12) levels in oral potentially malignant disorders (OPMDs), when observed may assist in early detection and prevention of these lesions. Hence the present study was conducted to assess and compare the plasma Vitamin B(12) levels in individuals with and without oral potentially malignant disorders. AIM: To assess and compare the plasma Vitamin B(12) levels in individuals with no oral lesions and individuals with oral potentially malignant disorders. MATERIALS AND METHODS: Systemically healthy individuals in the age group of 30 years-60 years of either gender with no oral lesions and those clinically diagnosed with oral potentially malignant disorders were selected for the study and grouped as Group A (30 individuals with no oral lesions) and Group B (30 individuals clinically diagnosed OPMDs). Plasma Vitamin B(12) levels were assessed using Electrochemiluminescence immunoassay. RESULTS: The Vitamin B(12) levels in Group A ranged between 224 and 913, while that in Group B ranged between 90 and 299; clearly indicating a decrease in the levels of Vitamin B(12) in individuals with OPMDs. CONCLUSION: Plasma Vitamin B(12) in individuals with oral potentially malignant disorders were decreased as compared to individuals with no oral lesions.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。