TRAUMATIC STRESS-RELATED ACCELERATED CELLULAR AGING

创伤应激相关的加速细胞衰老

阅读:1

Abstract

Recent research has identified a DNA methylation signature of biological aging. The “DNA methylation age” algorithms derived from this line of work can be used to determine the pathogenic factors that accelerate cellular aging (i.e., when cellular age exceeds chronological age) and the health correlates of advanced DNA methylation age. This presentation will review the results of several cross-sectional and longitudinal studies focused on traumatic-stress related accelerated aging and implications for health disparities. Results broadly suggest that posttraumatic stress disorder (PTSD) is associated with accelerated DNA methylation age and that accelerated DNA methylation age is associated with a variety of health conditions and outcomes, including metabolic syndrome, decreased neural integrity, and shortened time until death. Demographic factors may moderate these associations and help to explain age, sex, and/or race-related health disparities that are frequently reported in the literature.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。