The involvement of calmodulin and protein kinases in the upstream of cytosolic and nucleic calcium signaling induced by hypoosmotic shock in tobacco cells

钙调蛋白和蛋白激酶参与烟草细胞低渗冲击诱导的胞质和核钙信号传导的上游过程

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Abstract

Changes in Ca(2+) concentrations in cytosol ([Ca(2+)](C)) or nucleus ([Ca(2+)](N)) may play some vital roles in plants under hypoosmotic shock (Hypo-OS). Here, we observed that Hypo-OS induces biphasic increases in [Ca(2+)](C) and [Ca(2+)](N) in two tobacco cell lines (BY-2) expressing apoaequorin either in the cytosol or in the nucleus. Both [Ca(2+)](C) and [Ca(2+)](N) were sensitively modulated by the inhibitors of calmodulin and protein kinases, supporting the view that calmodulin suppresses the 1(st) peaks and and protein kinases enhance 2(nd) peaks in [Ca(2+)](C) and [Ca(2+)](N). Data also suggested that the 1(st) and 2(nd) events depend on the internal and extracellular Ca(2+) sources, respectively.

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