Aging impairs regulation of ryanodine receptors from extensor digitorum longus but not soleus muscles

衰老会损害趾长伸肌而非比目鱼肌中兰尼碱受体的调节。

阅读:1

Abstract

INTRODUCTION: Because impaired excitation-contraction coupling and reduced sarcoplasmic reticulum (SR) Ca(2+) release may contribute to the age-associated decline in skeletal muscle strength, we investigated the effect of aging on regulation of the skeletal muscle isoform of the ryanodine receptor (RyR1) by physiological channel ligands. METHODS: [(3) H]Ryanodine binding to membranes from 8- and 26-month-old Fischer 344 extensor digitorum longus (EDL) and soleus muscles was used to investigate the effects of age on RyR1 modulation by Ca(2+) and calmodulin (CaM). RESULTS: Aging reduced maximal Ca(2+) -stimulated binding to EDL membranes. In 0.3 μM Ca(2+) , age reduced binding and CaM increased binding to EDL membranes. In 300 μM Ca(2+) , CaM reduced binding, but the age effect was not significant. Aging did not affect Ca(2+) or CaM regulation of soleus RyR1. DISCUSSION: In aged fast-twitch muscle, impaired RyR1 Ca(2+) regulation may contribute to lower SR Ca(2+) release and reduced muscle function. Muscle Nerve 57: 1022-1025, 2018.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。