Hematin and Cu(II) control generations of hydroperoxyl and superoxide radicals to activate galactose oxidase for 5-hydroxymethylfurfural conversion

血红素和Cu(II)控制氢过氧自由基和超氧自由基的生成,从而激活半乳糖氧化酶,促进5-羟甲基糠醛的转化。

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Abstract

We present a significant finding that Cu(II) ions can activate hematin (Hem) to generate more HOO⋅ and O(2)(⋅-) radicals from the decomposition of H(2)O(2). Galactose oxidase (GO) and hematin have been simultaneously immobilized by coordinating to Cu(II) ions (GO&Hem@Cu(II)). The radicals HOO⋅ and O(2)(⋅-) and dioxygen O(2) can be in situ generated from the byproduct H(2)O(2) by the Cu(II)-activated hematin. Ample experimental evidence supports the discovery that the immobilized GO is reactivated by the in situ generated HOO⋅ and O(2)(⋅-). For the conversion of 100 mM 5-hydroxymethylfurfural (HMF) in water, GO&Hem@Cu(II) (0.8 mg/mL GO encapsulated) has achieved a 99.5% conversion within 180 min. In contrast, 0.8 mg/mL free GO M(3-5) variant (ACS Catalysis 2018, 8, 4025) has achieved an HMF conversion of 17.3%. For the conversion of HMF (1,000 mM) by GO&Hem@Cu(II) (4 mg/mL GO encapsulated), the HMF conversion is 98.8% after 8 h reaction.

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