Immunoexpression of programmed death receptor 1 and its ligands in oral cavity squamous cell carcinoma

口腔鳞状细胞癌中程序性死亡受体1及其配体的免疫表达

阅读:1

Abstract

BACKGROUND: This study analyzed the immunoexpression of programmed death receptor 1 and its ligands in oral cavity squamous cell carcinoma and correlated the findings with histomorphological parameters. MATERIAL AND METHODS: Forty cases of oral cavity squamous cell carcinoma (10 in the floor of the mouth, 10 in the palate, 10 in the lower lip, and 10 in the tongue) were selected. The percentages of cytoplasmic/membrane immunopositivity for programmed death receptor 1 and its ligands in neoplastic and stromal cells were evaluated at the tumor invasion front. RESULTS: Programmed death-ligand 1 presented high immunoexpression in all subsites, especially at the parenchyma level. Compared to floor of the mouth and palate, lower lip and tongue exhibited higher expression of programmed death receptor 1 and programmed death-ligand 2 in parenchymal cells and of programmed death receptor 1 in stromal cells, with statistically significant differences for programmed death receptor 1 expression in lower lip (p<0.05). Tongue presented the highest median percentages of positivity for programmed death-ligand 2, with statistically significant differences when compared to floor of the mouth (neoplastic cells and stromal cells) and lower lip (stromal cells) (p<0.05). Regarding histomorphological aspects, the inflammatory infiltrate appears to be an important factor for the immunoexpression of these proteins in oral cavity squamous cell carcinoma. CONCLUSIONS: The study suggests location-dependent differences in the antitumor immune response to oral cavity squamous cell carcinoma. Inflammatory infiltrate is key to protein immunoexpression. These findings are crucial for developing new immunotherapeutic strategies for oral cancer.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。