Pemafibrate as a therapeutic option for metabolic dysfunction-associated steatotic liver disease resistant to dietary intervention

培马贝特可作为治疗饮食干预无效的代谢功能障碍相关脂肪肝疾病的一种治疗选择

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Abstract

AIM OF THE STUDY: Metabolic dysfunction-associated steatotic liver disease (MASLD) is a lifestyle-related disease. However, many patients appear resistant to treatment despite dietary counseling. Pemafibrate is a novel lipidlowering pharmaceutical agent that was developed as a selective peroxisome proliferator-activated receptor α modulator. The present study aimed to clarify the efficacy of pemafibrate in diet-resistant MASLD patients with persistent hyperlipidemia. MATERIAL AND METHODS: Participants comprised 91 MASLD patients with persistent alanine aminotransferase (ALT) elevation and hyperlipidemia despite counseling on moderate-carbohydrate restriction (150-200 g/day) for more than one year, who received pemafibrate (0.2 mg/day) for 6 months. Complete response (CR) was defined as ALT normalization (≤ 33 IU/l in males, ≤ 25 IU/l in females). Partial response (PR) was defined as a ≥ 30% reduction in ALT level from baseline, without achieving CR. No response (NR) was defined as achieving neither CR nor PR. Dietary counseling was maintained every 3 months throughout pemafibrate treatment. RESULTS: CR, PR, and NR rates were 40% (36/91), 24% (22/91), and 36% (33/91), respectively. The response rate (CR + PR) was 64%. Although no significant change in body weight was observed, significant reductions were seen in lipid profiles (including triglycerides and low-density lipoprotein cholesterol) and liver enzymes (including ALT, aspartate aminotransferase, alkaline phosphatase, and γ-glutamyl transpeptidase). The concentration of glycosylated isomers of Mac-2-binding protein was significantly reduced, but no significant change in liver shear wave velocity was observed. CONCLUSIONS: Pemafibrate may be a potential second-line therapeutic option for diet-resistant MASLD patients with hyperlipidemia.

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