日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Insights into the structure and function of the RNA ligase RtcB

对RNA连接酶RtcB的结构和功能的新认识

Moncan, Matthieu; Rakhsh-Khorshid, Hassan; Eriksson, Leif A; Samali, Afshin; Gorman, Adrienne M

Endoplasmic reticulum stress induces ligand-independent TNFR1-mediated necroptosis in L929 cells

内质网应激诱导 L929 细胞发生配体非依赖性 TNFR1 介导的坏死性凋亡

S Saveljeva, S L Mc Laughlin, P Vandenabeele, A Samali, M J M Bertrand

Nerve growth factor-mediated inhibition of apoptosis post-caspase activation is due to removal of active caspase-3 in a lysosome-dependent manner

神经生长因子介导的 caspase 活化后细胞凋亡抑制是由于以溶酶体依赖的方式去除活性 caspase-3

K Mnich, L A Carleton, E T Kavanagh, K M Doyle, A Samali, A M Gorman

RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditions

在未解决的内质网应激条件下,RIPK1 促进死亡受体独立的 caspase-8 介导的细胞凋亡

Y Estornes, M A Aguileta, C Dubuisson, J De Keyser, V Goossens, K Kersse, A Samali, P Vandenabeele, M J M Bertrand

Resistance to TRAIL in non-transformed cells is due to multiple redundant pathways

非转化细胞对 TRAIL 的抗性是由于多个冗余途径

M van Dijk, A Halpin-McCormick, T Sessler, A Samali, E Szegezdi

Perk-dependent repression of miR-106b-25 cluster is required for ER stress-induced apoptosis

内质网应激诱导的细胞凋亡需要依赖 Perk 的 miR-106b-25 簇抑制

S Gupta, D E Read, A Deepti, K Cawley, A Gupta, D Oommen, T Verfaillie, S Matus, M A Smith, J L Mott, P Agostinis, C Hetz, A Samali

Rapid and efficient cancer cell killing mediated by high-affinity death receptor homotrimerizing TRAIL variants

高亲和力死亡受体同源三聚化TRAIL变体介导快速有效的癌细胞杀灭

C R Reis, A M van der Sloot, A Natoni, E Szegezdi, R Setroikromo, M Meijer, K Sjollema, F Stricher, R H Cool, A Samali, L Serrano, W J Quax

Nerve growth factor blocks thapsigargin-induced apoptosis at the level of the mitochondrion via regulation of Bim

神经生长因子通过调节 Bim 阻断线粒体水平的 thapsigargin 诱导的细胞凋亡

E Szegezdi, K Reed Herbert, E T Kavanagh, A Samali, A M Gorman