Alo Ray相关文献与解析，生知库 | 链接生命科学的每一步

Katie A Thies, Matthew W Cole, Rachel E Schafer, Jonathan M Spehar, Dillon S Richardson, Sarah A Steck, Manjusri Das, Arthur W Lian, Alo Ray, Reena Shakya, Sue E Knoblaugh, Cynthia D Timmers, Michael C Ostrowski, Arnab Chakravarti, Gina M Sizemore, Steven T Sizemore

肿瘤

乳腺癌

Mouse

发表日期：2021

文献求助收藏

ATR- and ATM-Mediated DNA Damage Response Is Dependent on Excision Repair Assembly during G1 but Not in S Phase of Cell Cycle

ATR 和 ATM 介导的 DNA 损伤反应依赖于细胞周期 G1 期而非 S 期的切除修复组装

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0159344

Alo Ray, Chessica Blevins, Gulzar Wani, Altaf A Wani

Induction of ATM/ATR pathway combined with Vγ2Vδ2 T cells enhances cytotoxicity of ovarian cancer cells

ATM/ATR通路诱导联合Vγ2Vδ2T细胞增强卵巢癌细胞的细胞毒作用

期刊:

影响因子:3.3

doi:10.1016/j.bbadis.2014.04.003

Jingwei Lu, Manjusri Das, Suman Kanji, Reeva Aggarwal, Matthew Joseph, Alo Ray, Charles L Shapiro, Vincent J Pompili, Hiranmoy Das

NER initiation factors, DDB2 and XPC, regulate UV radiation response by recruiting ATR and ATM kinases to DNA damage sites

NER 启动因子 DDB2 和 XPC 通过募集 ATR 和 ATM 激酶至 DNA 损伤位点来调节紫外线辐射反应

期刊:DNA Repair

影响因子:

doi:10.1016/j.dnarep.2013.01.003

Alo Ray, Keisha Milum, Aruna Battu, Gulzar Wani, Altaf A Wani

信号转导

发表日期：2013

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ASF1A and ATM regulate H3K56-mediated cell-cycle checkpoint recovery in response to UV irradiation

ASF1A 和 ATM 调节 H3K56 介导的细胞周期检查点恢复以响应紫外线照射

期刊:Nucleic Acids Research

影响因子:16.6

doi:10.1093/nar/gkr523

Aruna Battu, Alo Ray, Altaf A Wani

Human SNF5/INI1, a component of the human SWI/SNF chromatin remodeling complex, promotes nucleotide excision repair by influencing ATM recruitment and downstream H2AX phosphorylation

人类 SNF5/INI1 是人类 SWI/SNF 染色质重塑复合物的组成部分，它通过影响 ATM 募集和下游 H2AX 磷酸化来促进核苷酸切除修复

期刊:Molecular and Cellular Biology

影响因子:3.2

doi:10.1128/MCB.00503-09

Alo Ray, Safita N Mir, Gulzar Wani, Qun Zhao, Aruna Battu, Qianzheng Zhu, Qi-En Wang, Altaf A Wani

The small G-protein RalA promotes progression and metastasis of triple-negative breast cancer

小 G 蛋白 RalA 促进三阴性乳腺癌的进展和转移

ATR- and ATM-Mediated DNA Damage Response Is Dependent on Excision Repair Assembly during G1 but Not in S Phase of Cell Cycle

ATR 和 ATM 介导的 DNA 损伤反应依赖于细胞周期 G1 期而非 S 期的切除修复组装

Induction of ATM/ATR pathway combined with Vγ2Vδ2 T cells enhances cytotoxicity of ovarian cancer cells

ATM/ATR通路诱导联合Vγ2Vδ2T细胞增强卵巢癌细胞的细胞毒作用

NER initiation factors, DDB2 and XPC, regulate UV radiation response by recruiting ATR and ATM kinases to DNA damage sites

NER 启动因子 DDB2 和 XPC 通过募集 ATR 和 ATM 激酶至 DNA 损伤位点来调节紫外线辐射反应

ASF1A and ATM regulate H3K56-mediated cell-cycle checkpoint recovery in response to UV irradiation

ASF1A 和 ATM 调节 H3K56 介导的细胞周期检查点恢复以响应紫外线照射

Human SNF5/INI1, a component of the human SWI/SNF chromatin remodeling complex, promotes nucleotide excision repair by influencing ATM recruitment and downstream H2AX phosphorylation

人类 SNF5/INI1 是人类 SWI/SNF 染色质重塑复合物的组成部分，它通过影响 ATM 募集和下游 H2AX 磷酸化来促进核苷酸切除修复