日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Defective DNA Damage Response is a Targetable Therapeutic Vulnerability in ESR1 Mutant Breast Cancer.

DNA损伤反应缺陷是ESR1突变型乳腺癌的一个可靶向治疗弱点。

Herzog Sarah K, Stevens Jessica H, Gu Guowei, Grimm Sandra L, Cardoza Kloma, Hawkins Autumn G M, Lin Hangqing, Ramos Daniela, Beyer Amanda R, Edwards David G, Dustin Derek, Yang Harry J, Liang Nicole, Khan Ashfia F, Bawa-Khalfe Tasneem, McGrail Daniel J, Lin Shiaw-Yih, Coarfa Cristian, Fuqua Suzanne A W

Pathogenic variants in the cohesin loader subunit MAU2 underlie a distinct Cornelia de Lange Syndrome subtype.

黏连蛋白装载亚基 MAU2 的致病变异是 Cornelia de Lange 综合征的一个独特亚型的根本原因。

Parenti Ilaria, Hesters Alina, Gil-Salvador Marta, Duffy Laura, Kanber Deniz, Beygo Jasmin, Kerkhof Jennifer, Steenpaß Laura, Leitão Elsa, Woestefeld Julia, Boone Philip M, Kao Emeline M, Alabdi Lama, Aldhalaan Hesham M, Alkuraya Fowzan S, Alshammari Muneera J, Antonarakis Stylianos E, Basel Donald, Cassinari Kevin, de Polli Cellin Laurana, Clause Amanda R, de Lima Jorge Alexander Augusto, de Castro Leal Andréa, Collins Stephan C, Durand Benjamin, Eckhold Juliane, Hashem Mais O, Jayakar Parul, Khan Arif O, Kato Kohji, Kubica Regina, Lyon Gholson J, Marchi Elaine, McCarrier Julie, Kimmig Lara K, Mizuno Seiji, Nicolas Gael, Nishio Yosuke, Ogi Tomoo, Pié Juan, Prell Jordyn, Puisac Beatriz, Ramos Feliciano J, Ranza Emmanuelle, Redin Claire, Rush Eric, Saitoh Shinji, Shamseldin Hanan E, Starling Susan, Astiazaran-Symonds Esteban, Eltahir Sara H, Kuechler Alma, Sadikovic Bekim, Yalcin Binnaz, Wendt Kerstin S, Kaiser Frank J

Immunotherapy targeting drug-tolerant Mycobacterium tuberculosis persisters accelerates tuberculosis cure in preclinical models.

针对耐药结核分枝杆菌持续感染者的免疫疗法可加速临床前模型中结核病的治愈。

Karanika Styliani, Wang Tianyin, Yilma Addis, Ruelas Castillo Jennie, Gordy James T, Bailey Hannah, Quijada Darla, Fessler Kaitlyn, Tasneen Rokeya, Rouse Salcido Elisa M, Shamma Farah, Harris Harley T, Chen Fengyixin, Bates Rowan E, Ton Heemee, Meza Jacob, Li Yangchen, Taylor Alannah D, Zheng Jean J, Zhang Jiaqi, Karantanos Theodoros, Maxwell Amanda R, Nuermberger Eric, Peske J David, Markham Richard B, Karakousis Petros C

Donor-Recipient Age Mismatch and Long-Term Graft Outcomes After Adolescent Liver Transplant

供体-受体年龄不匹配与青少年肝移植术后长期移植物预后

Nakayama, Toshihiro; Jensen, Amanda R; Attia, Antony; Ahn, Daniel J; Firl, Daniel J; Kwong, Allison; Charu, Vivek; Melcher, Marc L; Esquivel, Carlos O; Sasaki, Kazunari

Lyme Disease and Health Care Costs

莱姆病与医疗保健成本

Yu, Holly; Fee, Rebecca M; Campfield, Brian T; Gould, L Hannah; Mercadante, Amanda R; Chastek, Benjamin; Johnson, Mary G; Bancroft, Tim; Halperin, John J

Risk Prediction Model for Taxane-Induced Peripheral Neuropathy in Early-Stage Cancer

早期癌症患者紫杉烷类药物诱发周围神经病变的风险预测模型

Trivedi, Meghna S; Unger, Joseph M; Henry, N Lynn; Darke, Amy K; Hertz, Daniel L; Brannagan, Thomas H; Reyes, Stephanie J; Schneider, Bryan P; Irvin, William J Jr; Hathaway, Amanda R; Vander Woude, Amy C; Gudena, Vinay K; Cabrera-Galeana, Paula; Orsted, Mary; LeBlanc, Michael; Fisch, Michael J; Hershman, Dawn L

Low Intensity Focused Ultrasound Neuromodulation in Psychiatric Disorders: Mechanisms, Models, and Missing Links

低强度聚焦超声神经调控在精神疾病中的应用:机制、模型和缺失环节

Arulpragasam, Amanda R; Theyel, Brian; Barredo, Jennifer; Pouille, Frederic; Gillotti, Julia G; Greenberg, Benjamin D; Wout-Frank, Mascha van 't; Philip, Noah S

Identification of Raptor and GLI1 as USP37 substrates highlight its context-specific function in medulloblastoma cells.

Raptor 和 GLI1 被鉴定为 USP37 的底物,突显了其在髓母细胞瘤细胞中的特定功能。

Singh Ashutosh, Cheng Donghang, Haltom Amanda R, Yang Yanwen, Dobson Tara, Hatcher Rashieda, Rajaram Veena, Gopalakrishnan Vidya

A quantitative cell-based reporter links TDP-43 aggregation and dysfunction to define pathogenic mechanisms

一种基于细胞的定量报告系统将TDP-43聚集和功能障碍联系起来,从而阐明其致病机制。

Mamede, Lohany Dias; Hu, Miwei; Vaquer-Alicea, Jaime; Titus, Amanda R; Passos, Patricia M; Lantelme, Erica; French, Rachel L; Kirschner, Paige A; Diamond, Marc I; Miller, Timothy M; Ayala, Yuna M

Elevated lactate in AML bone marrow contributes to macrophage polarization via GPR81 signaling

急性髓系白血病骨髓中乳酸水平升高通过GPR81信号通路促进巨噬细胞极化。

Soto, Celia A; Lesch, Maggie L; Becker, Jennifer L; Sharipol, Azmeer; Khan, Amal; Schafer, Xenia L; Li, Zhewen; Streeter, Amanda R; Becker, Michael W; Munger, Joshua C; Frisch, Benjamin J