Amaral Lorena M相关文献与解析，生知库 | 链接生命科学的每一步

Thomas Herrock, Owen; Campbell, Nathan; Deer, Evangeline; Amaral, Lorena M; Whitney, Darby; Morris, Rachael; Wallace, Kedra; Warren Turner, Ty; Cleveland, E Hawthorne; Belk, Sheila; Booz, George W; Cornelius, Denise C; LaMarca, Babbette

心血管

B细胞

免疫/内分泌

AT1-AA induced hypertension during pregnancy contributes to developmental programming of hypertension in female offspring

妊娠期AT1-AA诱导的高血压会导致雌性后代高血压的发育编程。

期刊:Journal of Reproductive Immunology

影响因子:

doi:10.1016/j.jri.2025.104802

Campbell, Nathan; Lemon, James P; Helmich, Melissa; Solise, Dylan; Demesa, Alexandra; Goolsby, Ella; LaMarca, Evan; Deer, Evangeline; Cornelius, Denise C; Turner, Ty; Amaral, Lorena M; Zheng, Baoying; LaMarca, Babbette

AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring Health

AT1-AA在胎盘缺血后于子代体内产生，B细胞耗竭可降低其水平，但不会影响子代的整体健康。

期刊:Journal of the American Heart Association

影响因子:5.3

doi:10.1161/JAHA.123.031417

Campbell, Nathan; Deer, Evangeline; Solise, Dylan; Cornelius, Denise C; Turner, Ty; Amaral, Lorena M; Herrock, Owen; Jordan, Ariel; Shukla, Shivani; Ibrahim, Tarek; LaMarca, Babbette

The role of immune cells and mediators in preeclampsia

免疫细胞和介质在先兆子痫中的作用

期刊:Nature Reviews Nephrology

影响因子:39.8

doi:10.1038/s41581-022-00670-0

Deer, Evangeline; Herrock, Owen; Campbell, Nathan; Cornelius, Denise; Fitzgerald, Sarah; Amaral, Lorena M; LaMarca, Babbette

RUPP Th17s cause hypertension and mitochondrial dysfunction in the kidney and placenta during pregnancy

RUPP Th17s 会导致妊娠期高血压以及肾脏和胎盘线粒体功能障碍。

期刊:Pregnancy Hypertension-An International Journal of Womens Cardiovascular Health

影响因子:2.9

doi:10.1016/j.preghy.2023.04.002

Fitzgerald, Sarah; Deer, Evangeline; Hogg, James; Cornelius, Denise C; Turner, Ty; Amaral, Lorena M; Hoang, Ngoc; Edwards, Kristin; Herrock, Owen; Campbell, Nathan; Ibrahim, Tarek; LaMarca, Babbette

VASCULAR AND RENAL MECHANISMS OF PREECLAMPSIA

子痫前期的血管和肾脏机制

期刊:

影响因子:

doi:10.1016/j.cophys.2023.100655

Wang, Xi; Shields, Corbin A; Ekperikpe, Ubong; Amaral, Lorena M; Williams, Jan Michael; Cornelius, Denise C

发表日期：2023

文献求助收藏

Investigation of interleukin-2-mediated changes in blood pressure, fetal growth restriction, and innate immune activation in normal pregnant rats and in a preclinical rat model of preeclampsia

研究白细胞介素-2介导的正常妊娠大鼠和先兆子痫临床前大鼠模型的血压变化、胎儿生长受限和先天免疫激活

期刊:Biology of Sex Differences

影响因子:5.1

doi:10.1186/s13293-020-00345-0

Cunningham, Mark W Jr; Amaral, Lorena M; Campbell, Nathan E; Cornelius, Denise C; Ibrahim, Tarek; Vaka, Venkata Ramana; LaMarca, Babbette

NAMPT single-nucleotide polymorphism rs1319501 and visfatin/NAMPT affect nitric oxide formation, sFlt-1 and antihypertensive therapy response in preeclampsia

NAMPT单核苷酸多态性rs1319501和visfatin/NAMPT影响先兆子痫患者的一氧化氮生成、sFlt-1和抗高血压治疗反应

期刊:Pharmacogenomics

影响因子:1.9

doi:10.2217/pgs-2021-0006

Pereira, Daniela A; Sandrim, Valeria C; Palei, Ana C; Amaral, Lorena M; Belo, Vanessa A; Lacchini, Riccardo; Cavalli, Ricardo C; Tanus-Santos, Jose E; Luizon, Marcelo R

Tumor necrosis factor alpha (TNF-α) blockade improves natural killer cell (NK) activation, hypertension, and mitochondrial oxidative stress in a preclinical rat model of preeclampsia

在先兆子痫的临床前大鼠模型中，肿瘤坏死因子α (TNF-α) 阻断可改善自然杀伤细胞 (NK) 活化、高血压和线粒体氧化应激。

期刊:Hypertension in Pregnancy

影响因子:2.1

doi:10.1080/10641955.2020.1793999

Cunningham, Mark W; Jayaram, Aswathi; Deer, Evangeline; Amaral, Lorena M; Vaka, Venkata Ramana; Ibrahim, Tarek; Cornelius, Denise C; LaMarca, Babbette

Placental CD4(+) T cells isolated from preeclamptic women cause preeclampsia-like symptoms in pregnant nude-athymic rats

从先兆子痫妇女体内分离出的胎盘CD4(+) T细胞可引起妊娠裸鼠出现类似先兆子痫的症状。

期刊:Pregnancy Hypertension-An International Journal of Womens Cardiovascular Health

影响因子:2.9

doi:10.1016/j.preghy.2018.10.007

Harmon, Ashlyn C; Ibrahim, Tarek; Cornelius, Denise C; Amaral, Lorena M; Cunningham, Mark W Jr; Wallace, Kedra; LaMarca, Babbette

Preeclamptic Placental CD19+ B Cells Are Causal to Hypertension During Pregnancy

子痫前期胎盘CD19+ B细胞是妊娠期高血压的病因

AT1-AA induced hypertension during pregnancy contributes to developmental programming of hypertension in female offspring

妊娠期AT1-AA诱导的高血压会导致雌性后代高血压的发育编程。

AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring Health

AT1-AA在胎盘缺血后于子代体内产生，B细胞耗竭可降低其水平，但不会影响子代的整体健康。

The role of immune cells and mediators in preeclampsia

免疫细胞和介质在先兆子痫中的作用

RUPP Th17s cause hypertension and mitochondrial dysfunction in the kidney and placenta during pregnancy

RUPP Th17s 会导致妊娠期高血压以及肾脏和胎盘线粒体功能障碍。

VASCULAR AND RENAL MECHANISMS OF PREECLAMPSIA

子痫前期的血管和肾脏机制

Investigation of interleukin-2-mediated changes in blood pressure, fetal growth restriction, and innate immune activation in normal pregnant rats and in a preclinical rat model of preeclampsia

研究白细胞介素-2介导的正常妊娠大鼠和先兆子痫临床前大鼠模型的血压变化、胎儿生长受限和先天免疫激活

NAMPT single-nucleotide polymorphism rs1319501 and visfatin/NAMPT affect nitric oxide formation, sFlt-1 and antihypertensive therapy response in preeclampsia

NAMPT单核苷酸多态性rs1319501和visfatin/NAMPT影响先兆子痫患者的一氧化氮生成、sFlt-1和抗高血压治疗反应

Tumor necrosis factor alpha (TNF-α) blockade improves natural killer cell (NK) activation, hypertension, and mitochondrial oxidative stress in a preclinical rat model of preeclampsia

在先兆子痫的临床前大鼠模型中，肿瘤坏死因子α (TNF-α) 阻断可改善自然杀伤细胞 (NK) 活化、高血压和线粒体氧化应激。

Placental CD4(+) T cells isolated from preeclamptic women cause preeclampsia-like symptoms in pregnant nude-athymic rats

从先兆子痫妇女体内分离出的胎盘CD4(+) T细胞可引起妊娠裸鼠出现类似先兆子痫的症状。