Misprogramming of glucose metabolism impairs recovery of hippocampal slices from neuronal GLT-1 knockout mice and contributes to excitotoxic injury through mitochondrial superoxide production.

葡萄糖代谢的错误编程会损害神经元 GLT-1 敲除小鼠海马切片的恢复，并通过线粒体超氧化物的产生导致兴奋性毒性损伤