Andreas Ivessa相关文献与解析，生知库 | 链接生命科学的每一步

Constitutive expression of cardiomyocyte Klf9 precipitates metabolic dysfunction and spontaneous cardiomyopathy.

心肌细胞 Klf9 的组成性表达会引发代谢功能障碍和自发性心肌病。

doi:10.1016/j.cellsig.2025.112146

Thakkar Chandni, Alikunju Saleena, Venkatasubramanian Aishwarya, D'Mello Dorothy, Abbas Hussain, Yang Zhi, Andreas Ivessa, Sayed Nazish, Abdellatif Maha, Sayed Danish

Bcl-x short-isoform is essential for maintaining homeostasis of multiple tissues

Bcl-x短亚型对于维持多种组织的稳态至关重要。

期刊:iScience

影响因子:4.6

doi:10.1016/j.isci.2023.106409

Mariko Aoyagi Keller,Chun-Yang Huang,Andreas Ivessa,Sukhwinder Singh,Peter J Romanienko,Michinari Nakamura

发表日期：2023

文献求助收藏

Ulk1-dependent alternative mitophagy plays a protective role during pressure overload in the heart

Ulk1 依赖的替代线粒体自噬在心脏压力超负荷期间起保护作用

期刊:Cardiovascular Research

影响因子:10.2

doi:10.1093/cvr/cvac003

Jihoon Nah, Akihiro Shirakabe, Risa Mukai, Peiyong Zhai, Eun Ah Sung, Andreas Ivessa, Wataru Mizushima, Yasuki Nakada, Toshiro Saito, Chengchen Hu, Yong Keun Jung, Junichi Sadoshima

Cathepsin D interacts with adenosine A2A receptors in mouse macrophages to modulate cell surface localization and inflammatory signaling

组织蛋白酶 D 与小鼠巨噬细胞中的腺苷 A2A 受体相互作用，调节细胞表面定位和炎症信号

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1016/j.jbc.2022.101888

Adrienn Skopál, Tamás Kéki, Péter Á Tóth, Balázs Csóka, Balázs Koscsó, Zoltán H Németh, Luca Antonioli, Andreas Ivessa, Francisco Ciruela, László Virág, György Haskó, Endre Kókai

Immunology/Inflammation

Mouse

发表日期：2022

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PERM1 regulates genes involved in fatty acid metabolism in the heart by interacting with PPARα and PGC-1α

PERM1 通过与 PPARα 和 PGC-1α 相互作用来调节参与心脏脂肪酸代谢的基因

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-022-18885-3

Chun-Yang Huang, Shin-Ichi Oka, Xiaoyong Xu, Chian-Feng Chen, Chien-Yi Tung, Ya-Yuan Chang, Youssef Mourad, Omair Vehra, Andreas Ivessa, Ghassan Yehia, Peter Romanienko, Chiao-Po Hsu, Junichi Sadoshima

代谢

发表日期：2022

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Dietary carbohydrates restriction inhibits the development of cardiac hypertrophy and heart failure

限制饮食中的碳水化合物可抑制心脏肥大和心力衰竭的发展

期刊:Cardiovascular Research

影响因子:10.2

doi:10.1093/cvr/cvaa298

Michinari Nakamura, Natalija Odanovic, Yasuki Nakada, Satomi Dohi, Peiyong Zhai, Andreas Ivessa, Zhi Yang, Maha Abdellatif, Junichi Sadoshima

AAV-mediated YAP expression in cardiac fibroblasts promotes inflammation and increases fibrosis

AAV介导的YAP在心脏成纤维细胞中的表达促进炎症并增加纤维化

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-021-89989-5

Jamie Francisco ，Yu Zhang ，Yasuki Nakada ，Jae Im Jeong ，Chun-Yang Huang ，Andreas Ivessa ，Shinichi Oka ，Gopal J Babu ，Dominic P Del Re

Mst1 inhibits autophagy by promoting the interaction between Beclin1 and Bcl-2

Mst1 通过促进 Beclin1 和 Bcl-2 之间的相互作用来抑制自噬

期刊:Nature Medicine

影响因子:58.7

doi:10.1038/nm.3322

Yasuhiro Maejima, Shiori Kyoi, Peiyong Zhai, Tong Liu, Hong Li, Andreas Ivessa, Sebastiano Sciarretta, Dominic P Del Re, Daniela K Zablocki, Chiao-Po Hsu, Dae-Sik Lim, Mitsuaki Isobe, Junichi Sadoshima

KAT6A acetylation regulates AMPK function and hypertrophic remodeling in the heart

KAT6A乙酰化调节AMPK功能和心脏肥大重塑

Constitutive expression of cardiomyocyte Klf9 precipitates metabolic dysfunction and spontaneous cardiomyopathy.

心肌细胞 Klf9 的组成性表达会引发代谢功能障碍和自发性心肌病。

Bcl-x short-isoform is essential for maintaining homeostasis of multiple tissues

Bcl-x短亚型对于维持多种组织的稳态至关重要。

Ulk1-dependent alternative mitophagy plays a protective role during pressure overload in the heart

Ulk1 依赖的替代线粒体自噬在心脏压力超负荷期间起保护作用

Cathepsin D interacts with adenosine A2A receptors in mouse macrophages to modulate cell surface localization and inflammatory signaling

组织蛋白酶 D 与小鼠巨噬细胞中的腺苷 A2A 受体相互作用，调节细胞表面定位和炎症信号

PERM1 regulates genes involved in fatty acid metabolism in the heart by interacting with PPARα and PGC-1α

PERM1 通过与 PPARα 和 PGC-1α 相互作用来调节参与心脏脂肪酸代谢的基因

Dietary carbohydrates restriction inhibits the development of cardiac hypertrophy and heart failure

限制饮食中的碳水化合物可抑制心脏肥大和心力衰竭的发展

AAV-mediated YAP expression in cardiac fibroblasts promotes inflammation and increases fibrosis

AAV介导的YAP在心脏成纤维细胞中的表达促进炎症并增加纤维化

Mst1 inhibits autophagy by promoting the interaction between Beclin1 and Bcl-2

Mst1 通过促进 Beclin1 和 Bcl-2 之间的相互作用来抑制自噬