日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

miRNA/mRNA analysis of increased TGF-β pathways drive epithelial-mesenchymal transition and regulatory T cell differentiation

miRNA/mRNA分析表明,TGF-β通路增强可驱动上皮-间质转化和调节性T细胞分化。

Darville, Toni; Sun, Xuejun; Zhang, Yu; O'Connell, Catherine M; Mokashi, Neha; Tang, Weiming; Bhardwaj, Aakash; Duncan, Bryce; Andrews, Charles W Jr; Weisenfeld, Harold; Zheng, Xiaojing

Steroids alone or as adjunctive therapy with doxycycline fail to improve oviduct damage in mice infected with Chlamydia muridarum

单独使用类固醇或与强力霉素联合使用均未能改善感染鼠衣原体的鼠的输卵管损伤。

Corr, Tammy E; Sullivan, Jeanne; Frazer, Lauren C; Andrews, Charles W Jr; O'Connell, Catherine M; Darville, Toni

IL-23 induces IL-22 and IL-17 production in response to Chlamydia muridarum genital tract infection, but the absence of these cytokines does not influence disease pathogenesis.

IL-23 可诱导 IL-22 和 IL-17 的产生以应对鼠衣原体生殖道感染,但这些细胞因子的缺失并不影响疾病的发病机制

Frazer Lauren C, Scurlock Amy M, Zurenski Matthew A, Riley Melissa M, Mintus Margaret, Pociask Derek A, Sullivan Jeanne E, Andrews Charles W Jr, Darville Toni

Plasmid-cured Chlamydia caviae activates TLR2-dependent signaling and retains virulence in the guinea pig model of genital tract infection

经质粒清除的豚鼠衣原体可激活 TLR2 依赖性信号通路,并在豚鼠生殖道感染模型中保留毒力。

Frazer, Lauren C; Darville, Toni; Chandra-Kuntal, Kumar; Andrews, Charles W Jr; Zurenski, Matthew; Mintus, Margaret; AbdelRahman, Yasser M; Belland, Robert J; Ingalls, Robin R; O'Connell, Catherine M

The recall response induced by genital challenge with Chlamydia muridarum protects the oviduct from pathology but not from reinfection

用鼠衣原体进行生殖器感染后诱发的记忆性免疫反应可以保护输卵管免受病变,但不能防止再次感染。

Riley, Melissa M; Zurenski, Matthew A; Frazer, Lauren C; O'Connell, Catherine M; Andrews, Charles W Jr; Mintus, Margaret; Darville, Toni

MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resolution

MyD88 缺陷会导致鼠衣原体生殖道感染期间 NK 细胞 γ 干扰素产生减少和 T 细胞募集减少,但以 Th1 反应为主和单核细胞炎症增强与感染消退相关。

Nagarajan, Uma M; Sikes, James; Prantner, Daniel; Andrews, Charles W Jr; Frazer, Lauren; Goodwin, Anna; Snowden, Jessica N; Darville, Toni

Interleukin-17 contributes to generation of Th1 immunity and neutrophil recruitment during Chlamydia muridarum genital tract infection but is not required for macrophage influx or normal resolution of infection

白细胞介素-17有助于在鼠衣原体生殖道感染期间产生Th1免疫反应和中性粒细胞募集,但并非巨噬细胞浸润或感染正常消退所必需。

Scurlock, Amy M; Frazer, Lauren C; Andrews, Charles W Jr; O'Connell, Catherine M; Foote, Isaac P; Bailey, Sarabeth L; Chandra-Kuntal, Kumar; Kolls, Jay K; Darville, Toni

Infectivity acts as in vivo selection for maintenance of the chlamydial cryptic plasmid

感染性在体内起到维持衣原体隐蔽质粒的选择作用

Russell, Marsha; Darville, Toni; Chandra-Kuntal, Kumar; Smith, Bennett; Andrews, Charles W Jr; O'Connell, Catherine M

Enhanced neutrophil longevity and recruitment contribute to the severity of oviduct pathology during Chlamydia muridarum infection.

中性粒细胞寿命延长和募集增加会导致鼠衣原体感染期间输卵管病变的严重程度增加

Frazer Lauren C, O'Connell Catherine M, Andrews Charles W Jr, Zurenski Matthew A, Darville Toni

Type I interferon signaling exacerbates Chlamydia muridarum genital infection in a murine model

I型干扰素信号传导会加剧小鼠模型中的鼠衣原体生殖器感染

Nagarajan, Uma M; Prantner, Daniel; Sikes, James D; Andrews, Charles W Jr; Goodwin, Anna M; Nagarajan, Shanmugam; Darville, Toni