Anna Fracassi相关文献与解析，生知库 | 链接生命科学的每一步

Annalisa, Bernareggi; Ru, Zhang Wen; Laura, Sanchez-Sanchez; Alessia, Norbedo; Anna, Fracassi; Marianna, Lucafò; Marina, Sciancalepore; Griffoni, Alberto; William, Russell K; Giulio, Taglialatela; Paola, Lorenzon; Agenor, Limon

ZO1

细胞生物学

囊泡

发表日期：2026

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Sub-Immunosuppressive Tacrolimus Ameliorates Amyloid-Beta and Tau Pathology in 3xTg-AD Mice

亚免疫抑制他克莫司可改善 3xTg-AD 小鼠的淀粉样蛋白 β 和 Tau 病理

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms26051797

Jacqueline Silva, Batbayar Tumurbaatar, Jutatip Guptarak, Wen-Ru Zhang, Anna Fracassi, Giulio Taglialatela

免疫

发表日期：2025

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Cognitive integrity in Non-Demented Individuals with Alzheimer's Neuropathology is associated with preservation and remodeling of dendritic spines

患有阿尔茨海默病神经病理学的非痴呆患者的认知完整性与树突棘的保存和重塑有关

期刊:Alzheimers & Dementia

影响因子:13

doi:10.1002/alz.13900

Jutatip Guptarak, Pietro Scaduto, Batbayar Tumurbaatar, Wen Ru Zhang, Daniel Jupiter, Giulio Taglialatela, Anna Fracassi

Amyloid-β oligomers increase the binding and internalization of tau oligomers in human synapses

β-淀粉样蛋白寡聚体增加人类突触中tau蛋白寡聚体的结合和内化

期刊:Acta Neuropathologica

影响因子:9.3

doi:10.1007/s00401-024-02839-2

Shrinath Kadamangudi ，Michela Marcatti ，Wen-Ru Zhang ，Anna Fracassi ，Rakez Kayed ，Agenor Limon ，Giulio Taglialatela

神经科学

发表日期：2024

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Arbovirus infection increases the risk for the development of neurodegenerative disease pathology in the murine model

虫媒病毒感染增加了小鼠模型中神经退行性疾病病理发展的风险

期刊:Brain Behavior Immunity Health

影响因子:3.7

doi:10.1016/j.bbih.2024.100780

Chanida Fongsaran, Krit Jirakanwisal, Bi-Hung Peng, Anna Fracassi, Giulio Taglialatela, Kelly T Dineley, Slobodan Paessler, Irma E Cisneros

神经

发表日期：2024

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Inhibition of Calcineurin with FK506 Reduces Tau Levels and Attenuates Synaptic Impairment Driven by Tau Oligomers in the Hippocampus of Male Mouse Models

用FK506抑制钙调磷酸酶可降低雄性小鼠海马中Tau蛋白水平并减轻Tau蛋白寡聚体引起的突触功能障碍

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms25169092

Michela Marcatti ，Batbayar Tumurbaatar ，Michela Borghi ，Jutatip Guptarak ，Wen-Ru Zhang ，Balaji Krishnan ，Rakez Kayed ，Anna Fracassi ，Giulio Taglialatela

Preserved autophagy in cognitively intact non-demented individuals with Alzheimer's neuropathology

认知完整的非痴呆阿尔茨海默病神经病理患者的自噬得以保留

期刊:Alzheimers & Dementia

影响因子:13

doi:10.1002/alz.13074

Batbayar Tumurbaatar, Anna Fracassi, Pietro Scaduto, Jutatip Guptarak, Randall Woltjer, Daniel Jupiter, Giulio Taglialatela

Functional excitatory to inhibitory synaptic imbalance and loss of cognitive performance in people with Alzheimer's disease neuropathologic change

阿尔茨海默病患者的功能性兴奋性与抑制性突触失衡和认知能力丧失的神经病理学变化

期刊:Acta Neuropathologica

影响因子:9.3

doi:10.1007/s00401-022-02526-0

Pietro Scaduto, Julie C Lauterborn, Conor D Cox, Anna Fracassi, Tommaso Zeppillo, Berenice A Gutierrez, C Dirk Keene, Paul K Crane, Shubhabrata Mukherjee, William K Russell, Giulio Taglialatela, Agenor Limon

TREM2-induced activation of microglia contributes to synaptic integrity in cognitively intact aged individuals with Alzheimer's neuropathology

TREM2诱导的小胶质细胞活化有助于维持认知功能正常的阿尔茨海默病老年患者的突触完整性。

期刊:Brain Pathology

影响因子:5.8

doi:10.1111/bpa.13108

Anna Fracassi ，Michela Marcatti ，Batbayar Tumurbaatar ，Randall Woltjer ，Sandra Moreno ，Giulio Taglialatela

Aβ/tau oligomer interplay at human synapses supports shifting therapeutic targets for Alzheimer's disease

人类突触中Aβ/tau寡聚体的相互作用支持阿尔茨海默病治疗靶点的转变

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-022-04255-9

Michela Marcatti ，Anna Fracassi ，Mauro Montalbano ，Chandramouli Natarajan ，Balaji Krishnan ，Rakez Kayed ，Giulio Taglialatela

PIEZO1 Channels Modulate the Small Extracellular Vesicle Release in C2C12 Cells

PIEZO1通道调节C2C12细胞中小细胞外囊泡的释放

Sub-Immunosuppressive Tacrolimus Ameliorates Amyloid-Beta and Tau Pathology in 3xTg-AD Mice

亚免疫抑制他克莫司可改善 3xTg-AD 小鼠的淀粉样蛋白 β 和 Tau 病理

Cognitive integrity in Non-Demented Individuals with Alzheimer's Neuropathology is associated with preservation and remodeling of dendritic spines

患有阿尔茨海默病神经病理学的非痴呆患者的认知完整性与树突棘的保存和重塑有关

Amyloid-β oligomers increase the binding and internalization of tau oligomers in human synapses

β-淀粉样蛋白寡聚体增加人类突触中tau蛋白寡聚体的结合和内化

Arbovirus infection increases the risk for the development of neurodegenerative disease pathology in the murine model

虫媒病毒感染增加了小鼠模型中神经退行性疾病病理发展的风险

Inhibition of Calcineurin with FK506 Reduces Tau Levels and Attenuates Synaptic Impairment Driven by Tau Oligomers in the Hippocampus of Male Mouse Models

用FK506抑制钙调磷酸酶可降低雄性小鼠海马中Tau蛋白水平并减轻Tau蛋白寡聚体引起的突触功能障碍

Preserved autophagy in cognitively intact non-demented individuals with Alzheimer's neuropathology

认知完整的非痴呆阿尔茨海默病神经病理患者的自噬得以保留

Functional excitatory to inhibitory synaptic imbalance and loss of cognitive performance in people with Alzheimer's disease neuropathologic change

阿尔茨海默病患者的功能性兴奋性与抑制性突触失衡和认知能力丧失的神经病理学变化

TREM2-induced activation of microglia contributes to synaptic integrity in cognitively intact aged individuals with Alzheimer's neuropathology

TREM2诱导的小胶质细胞活化有助于维持认知功能正常的阿尔茨海默病老年患者的突触完整性。

Aβ/tau oligomer interplay at human synapses supports shifting therapeutic targets for Alzheimer's disease

人类突触中Aβ/tau寡聚体的相互作用支持阿尔茨海默病治疗靶点的转变