日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Casz1 and Znf101/Zfp961 differentially regulate apolipoproteins A1 and B, alter plasma lipoproteins, and reduce atherosclerosis

Casz1 和 Znf101/Zfp961 差异性地调节载脂蛋白 A1 和 B,改变血浆脂蛋白,并减少动脉粥样硬化

Ansari, Abulaish; Yadav, Pradeep Kumar; Zhou, Liye; Prakash, Binu; Ijaz, Laraib; Christiano, Amanda; Ahmad, Sameer; Rimbert, Antoine; Hussain, M Mahmood

Human hepatoma Huh-7 cell culture models deficient in apolipoprotein B secretion.

人肝癌 Huh-7 细胞培养模型缺乏载脂蛋白 B 分泌

Valmiki Swati, Rosario Sara, Mooring Ashley, Prakashmurthy Chandana, Schlamp Florencia, Prakash Binu, Chattopadhyay Atrayee, Ansari Abulaish, Alemán José O, Pamir Nathalie, Hussain M Mahmood

MicroRNA-541-3p alters lipoproteins to reduce atherosclerosis by degrading Znf101 and Casz1 transcription factors

MicroRNA-541-3p通过降解Znf101和Casz1转录因子来改变脂蛋白,从而减少动脉粥样硬化。

Ansari, Abulaish; Yadav, Pradeep Kumar; Zhou, Liye; Prakash, Binu; Gangula, Bhargavi; Ijaz, Laraib; Christiano, Amanda; Ahmad, Sameer; Rimbert, Antoine; Hussain, M Mahmood

Genetically engineered synthetic miniaturized versions of Plasmodium falciparum UvrD helicase are catalytically active

经基因工程改造的合成微型化恶性疟原虫UvrD解旋酶具有催化活性。

Ansari, Abulaish; Tarique, Mohammed; Tuteja, Renu

Plasmodium falciparum UvrD helicase translocates in 3' to 5' direction, colocalizes with MLH and modulates its activity through physical interaction

恶性疟原虫UvrD解旋酶沿3'至5'方向易位,与MLH共定位,并通过物理相互作用调节其活性。

Ahmad, Moaz; Ansari, Abulaish; Tarique, Mohammed; Satsangi, Akash Tripathi; Tuteja, Renu

Genome wide comparative comprehensive analysis of Plasmodium falciparum MCM family with human host

对恶性疟原虫MCM家族与人类宿主进行全基因组比较综合分析

Ansari, Abulaish; Tuteja, Renu

Emerging functions of transcription factors in malaria parasite

疟原虫转录因子的新兴功能

Tuteja, Renu; Ansari, Abulaish; Chauhan, Virander Singh