日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

ATP1A3 dysfunction causes motor hyperexcitability and afterhyperpolarization loss in a dystonia model

ATP1A3功能障碍导致肌张力障碍模型中运动过度兴奋和后超极化丧失

Akkuratov, Evgeny E; Sorrell, Francesca; Picton, Laurence D; Sousa, Vasco C; Paucar, Martin; Jans, Daniel; Svensson, Lill-Britt; Lindskog, Maria; Fritz, Nicolas; Liebmann, Thomas; Sillar, Keith T; Rosewich, Hendrik; Svenningsson, Per; Brismar, Hjalmar; Miles, Gareth B; Aperia, Anita

An interaction between PRRT2 and Na+/K+ ATPase contributes to the control of neuronal excitability

PRRT2 与 Na+/K+ ATPase 之间的相互作用有助于控制神经元兴奋性

Bruno Sterlini #, Alessandra Romei #, Chiara Parodi, Davide Aprile, Michele Oneto, Anita Aperia, Pierluigi Valente, Flavia Valtorta, Anna Fassio, Pietro Baldelli, Fabio Benfenati #, Anna Corradi #

Super-resolution microscopy reveals that Na+/K+-ATPase signaling protects against glucose-induced apoptosis by deactivating Bad

超分辨率显微镜显示,Na+/K+-ATPase信号通路通过抑制Bad蛋白的活性来抵抗葡萄糖诱导的细胞凋亡。

Kristoffer Bernhem # ,Jacopo M Fontana # ,Daniel Svensson # ,Liang Zhang # ,Linnéa M Nilsson ,Lena Scott ,Hans Blom ,Hjalmar Brismar ,Anita Aperia

A missense mutation converts the Na+,K+-ATPase into an ion channel and causes therapy-resistant epilepsy

错义突变将 Na+、K+-ATPase 转化为离子通道,并导致难治性癫痫

Sofia Ygberg, Evgeny E Akkuratov, Rebecca J Howard, Fulya Taylan, Daniel C Jans, Dhani R Mahato, Adriana Katz, Paula F Kinoshita, Benjamin Portal, Inger Nennesmo, Maria Lindskog, Steven J D Karlish, Magnus Andersson, Anna Lindstrand, Hjalmar Brismar, Anita Aperia

Ouabain Modulates the Functional Interaction Between Na,K-ATPase and NMDA Receptor

乌本苷调节Na,K-ATP酶与NMDA受体之间的功能性相互作用

Akkuratov, Evgeny E; Westin, Linda; Vazquez-Juarez, Erika; de Marothy, Minttu; Melnikova, Aleksandra K; Blom, Hans; Lindskog, Maria; Brismar, Hjalmar; Aperia, Anita

Ouabain-regulated phosphoproteome reveals molecular mechanisms for Na+, K+-ATPase control of cell adhesion, proliferation, and survival

乌巴因调节的磷酸化蛋白质组揭示了 Na+、K+-ATPase 控制细胞粘附、增殖和存活的分子机制

Elena Panizza, Liang Zhang, Jacopo Maria Fontana, Kozo Hamada, Daniel Svensson, Evgeny E Akkuratov, Lena Scott, Katsuhiko Mikoshiba, Hjalmar Brismar, Janne Lehtiö, Anita Aperia

Spontaneous calcium activity in metanephric mesenchymal cells regulates branching morphogenesis in the embryonic kidney

后肾间充质细胞中的自发性钙活动调节胚胎肾脏的分支形态发生

Fontana, Jacopo M; Khodus, Georgiy R; Unnersjö-Jess, David; Blom, Hans; Aperia, Anita; Brismar, Hjalmar

Prompt apoptotic response to high glucose in SGLT-expressing renal cells

SGLT 表达肾细胞对高葡萄糖的迅速凋亡反应

Linnéa M Nilsson, Liang Zhang, Alexander Bondar, Daniel Svensson, Annika Wernerson, Hjalmar Brismar, Lena Scott, Anita Aperia

Regulation of Neuronal Na,K-ATPase by Extracellular Scaffolding Proteins

细胞外支架蛋白对神经元Na,K-ATP酶的调控

Thomas Liebmann ,Nicolas Fritz ,Markus Kruusmägi ,Linda Westin ,Kristoffer Bernhem ,Alexander Bondar ,Anita Aperia ,Hjalmar Brismar

AT1-receptor response to non-saturating Ang-II concentrations is amplified by calcium channel blockers

钙通道阻滞剂可增强 AT1 受体对非饱和血管紧张素 II 浓度的反应

Kristoffer Bernhem, Kalaiselvan Krishnan, Alexander Bondar, Hjalmar Brismar, Anita Aperia, Lena Scott