日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Context-Dependent Roles of ANGPTL2-Mediated Inflammaging in Tissue Homeostasis, Pathological Tissue Remodeling, and Longevity.

ANGPTL2 介导的炎症衰老在组织稳态、病理组织重塑和寿命中发挥着依赖于上下文的作用。

Yumoto Shinsei, Horiguchi Haruki, Miyata Keishi, Kadomatsu Tsuyoshi, Tian Zhe, Sato Michio, Araki Kimi, Iwatsuki Masaaki, Oike Yuichi

Machine learning-assisted decoding of temporal transcriptional dynamics via fluorescent timer

利用荧光计时器进行机器学习辅助的时间转录动态解码

Irie, Nobuko; Takeda, Naoki; Satou, Yorifumi; Araki, Kimi; Ono, Masahiro

GALNTL5 binds GalNAc and is required for migration through the uterotubal junction and sperm-zona pellucida binding

GALNTL5 结合 GalNAc,是精子通过输卵管子宫连接处以及与透明带结合所必需的。

Noda, Taichi; Uriu, Reika; Mashiko, Daisuke; Shinohara, Hina; Qu, Yongcun; Taira, Ayumu; Matzuk, Ryan M; Tahala, Duri; Nakano, Motochika; Araki, Kimi; Yu, Zhifeng; Zhang, Ying; Matzuk, Martin M; Ikawa, Masahito

Regulation of H3K4me3 breadth and MYC expression by the SETD1B catalytic domain in MLL-rearranged leukemia

MLL重排白血病中SETD1B催化结构域对H3K4me3广度和MYC表达的调控

Izumi, Shintaro; Ohtani, Ko; Matsumoto, Makoto; Shibata, Seito; Rahmutulla, Bahityar; Fukuyo, Masaki; Nishimoto, Mitsutaka; Miyagawa, Hideo; Sakaida, Emiko; Yokote, Koutaro; Kitabayashi, Issay; Araki, Kimi; Kaneda, Atsushi; Hoshii, Takayuki

Extrinsic induction of apoptosis and tumor suppression via the p53-Reprimo-Hippo-YAP/TAZ-p73 pathway.

通过 p53-Reprimo-Hippo-YAP/TAZ-p73 通路诱导细胞凋亡和肿瘤抑制

Takikawa Masahiro, Nakano Airi, Krishnaraj Jayaraman, Tabata Yuko, Watanabe Yuzo, Okabe Atsushi, Sakaguchi Yukiko, Fujiki Ryoji, Mochizuki Ami, Tajima Tomoko, Sada Akane, Matsushita Shu, Wakabayashi Yuichi, Araki Kimi, Kaneda Atsushi, Ishikawa Fuyuki, Sadaie Mahito, Ohki Rieko

Mice with Heterozygous Deletion of Exon 3 in the Gh Gene Demonstrate Growth Retardation Caused by Reduced Ghrhr mRNA

Gh基因外显子3杂合缺失的小鼠表现出生长迟缓,这是由于Ghrhr mRNA减少所致。

Ariyasu, Daisuke; Higa, Daisuke; Tokudome, Ryo; Yonemori, Takumi; Shimada, Hayate; Shibata, Shinsuke; Araki, Kimi

TREATMENT FOR ATTENTION-DEFICIT HYPERACTIVITY DISORDER (ADHD) DRUG NORMALIZES AIS LENGTH ABNORMALITY ALONG WITH HYPERACTIVITY IN PITUITARY ADENYLATE CYCLASE-ACTIVATING POLYPEPTIDE (PACAP)-DEFICIENT MICE

治疗注意力缺陷多动障碍(ADHD)的药物可使垂体腺苷酸环化酶激活多肽(PACAP)缺陷小鼠的轴突起始段(AIS)长度异常以及多动症恢复正常

Ong, Jun; Sasaki, Kazunori; Ferdousi, Farhana; Suresh, Megalakshmi; Isoda, Hiroko; Szele, Francis G; Wenbing, *Chen; Takashi, Asano; Notaka, Izuo; Shin-Ichi, Muramatsu; Atsumi, Nitta; Kajitani, *Naoto; Okada-Tsuchioka, Mami; Inoue, Asuka; Miyano, Kanako; Masuda, Takeshi; Boku, Shuken; Iwamoto, Kazuya; Ohtsuki, Sumio; Uezono, Yasuhito; Aoki, Junken; Takebayashi, Minoru; Victoriano, *Gerardo Mendez; Zhu, Yunting; Middleton, Frank; Webster, Maree J; Weickert, Cynthia S; Guo, *Chu; Fujii, Shinya; Imamura, Yuko; Akamine, Keiya; Bundo, Miki; Nakachi, Yutaka; Araki, Kimi; Iwamoto, Kazuya; Iwahashi, *Misaki; Yoshimura, Takeshi; Harigai, Wakana; Takuma, Kazuhiro; Hashimoto, Hitoshi; Katayama, Taiichi; Hayata-Takano, Atsuko

Multiple genes in the Pate5-13 genomic region contribute to ADAM3 processing†

Pate5-13基因组区域中的多个基因参与ADAM3的加工†

Noda, Taichi; Shinohara, Hina; Kobayashi, Sumire; Taira, Ayumu; Oura, Seiya; Tahara, Duri; Tokuyasu, Midori; Araki, Kimi; Ikawa, Masahito

Multiple myeloma-associated DIS3 gene is essential for hematopoiesis, but loss of DIS3 is insufficient for myelomagenesis

多发性骨髓瘤相关的DIS3基因对造血至关重要,但DIS3的缺失并不足以导致骨髓瘤的发生。

Ohguchi, Hiroto; Ohguchi, Yasuyo; Kubota, Sho; Etoh, Kan; Hamashima, Ai; Usuki, Shingo; Yokomizo-Nakano, Takako; Bai, Jie; Masuda, Takeshi; Kawano, Yawara; Harada, Takeshi; Nakao, Mitsuyoshi; Minami, Takashi; Hideshima, Teru; Araki, Kimi; Sashida, Goro

A cyclic pyrrole-imidazole polyamide reduces pathogenic RNA in CAG/CTG triplet repeat neurological disease models

环状吡咯-咪唑聚酰胺可减少CAG/CTG三核苷酸重复神经系统疾病模型中的致病性RNA

Ikenoshita, Susumu; Matsuo, Kazuya; Yabuki, Yasushi; Kawakubo, Kosuke; Asamitsu, Sefan; Hori, Karin; Usuki, Shingo; Hirose, Yuki; Bando, Toshikazu; Araki, Kimi; Ueda, Mitsuharu; Sugiyama, Hiroshi; Shioda, Norifumi