Long-term immune reprogramming of classical monocytes with altered ontogeny mediates enhanced lung injury in sepsis survivors.
经典单核细胞的长期免疫重编程及其个体发育的改变,导致脓毒症幸存者肺损伤加剧
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doi:10.1101/2025.05.16.654442
Denstaedt Scott J, McBean Breanna, Boyle Alan P, Arenberg Brett C, Mack Matthias, Moore Bethany B, Newstead Michael W, Deng Yanmei, Nesvizhskii Alexey I, Singer Benjamin H, Cano Jennifer, Prescott Hallie C, Goodridge Helen S, Zemans Rachel L