Armengod相关文献与解析，生知库 | 链接生命科学的每一步

The MELAS mutation m.3243A>G promotes reactivation of fetal cardiac genes and an epithelial-mesenchymal transition-like program via dysregulation of miRNAs

MELAS 突变 m.3243A>G 通过 miRNA 失调促进胎儿心脏基因的重新激活和上皮-间质转化样程序

期刊:Biochimica et Biophysica Acta-Molecular Basis of Disease

doi:10.1016/j.bbadis.2018.06.014

Salvador Meseguer, Joaquin Panadero, Carmen Navarro-González, Magda Villarroya, Rachid Boutoual, Giacomo Pietro Comi, M-Eugenia Armengod

信号转导

发表日期：2018

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Defects in the mitochondrial-tRNA modification enzymes MTO1 and GTPBP3 promote different metabolic reprogramming through a HIF-PPARγ-UCP2-AMPK axis

线粒体 tRNA 修饰酶 MTO1 和 GTPBP3 的缺陷通过 HIF-PPARγ-UCP2-AMPK 轴促进不同的代谢重编程

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-018-19587-5

Rachid Boutoual, Salvador Meseguer, Magda Villarroya, Elena Martín-Hernández, Mohammed Errami, Miguel A Martín, Marta Casado, M-Eugenia Armengod

代谢

发表日期：2018

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microRNA-mediated differential expression of TRMU, GTPBP3 and MTO1 in cell models of mitochondrial-DNA diseases

microRNA 介导的线粒体 DNA 疾病细胞模型中 TRMU、GTPBP3 和 MTO1 的差异表达

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-017-06553-w

Salvador Meseguer, Olga Boix, Carmen Navarro-González, Magda Villarroya, Rachid Boutoual, Sonia Emperador, Elena García-Arumí, Julio Montoya, M-Eugenia Armengod

Mutations in the Caenorhabditis elegans orthologs of human genes required for mitochondrial tRNA modification cause similar electron transport chain defects but different nuclear responses

线粒体 tRNA 修饰所需的人类基因的秀丽隐杆线虫直系同源物的突变会导致类似的电子传递链缺陷，但会导致不同的核反应

期刊:PLoS Genetics

影响因子:

doi:10.1371/journal.pgen.1006921

Carmen Navarro-González, Ismaïl Moukadiri, Magda Villarroya, Ernesto López-Pascual, Simon Tuck, M-Eugenia Armengod

The ROS-sensitive microRNA-9/9* controls the expression of mitochondrial tRNA-modifying enzymes and is involved in the molecular mechanism of MELAS syndrome

ROS 敏感的 microRNA-9/9* 控制线粒体 tRNA 修饰酶的表达，并参与 MELAS 综合征的分子机制

期刊:Human Molecular Genetics

影响因子:3.1

doi:10.1093/hmg/ddu427

Salvador Meseguer, Ana Martínez-Zamora, Elena García-Arumí, Antonio L Andreu, M-Eugenia Armengod

信号转导

发表日期：2015

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Defective Expression of the Mitochondrial-tRNA Modifying Enzyme GTPBP3 Triggers AMPK-Mediated Adaptive Responses Involving Complex I Assembly Factors, Uncoupling Protein 2, and the Mitochondrial Pyruvate Carrier

线粒体 tRNA 修饰酶 GTPBP3 表达缺陷会触发 AMPK 介导的适应性反应，涉及复合物 I 组装因子、解偶联蛋白 2 和线粒体丙酮酸载体

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0144273

Ana Martínez-Zamora, Salvador Meseguer, Juan M Esteve, Magda Villarroya, Carmen Aguado, J Antonio Enríquez, Erwin Knecht, M-Eugenia Armengod

Intrapartum synthetic oxytocin reduce the expression of primitive reflexes associated with breastfeeding

产时合成催产素会降低与母乳喂养相关的原始反射的表达。

期刊:Breastfeeding Medicine

影响因子:1.8

doi:10.1089/bfm.2014.0156

Marín Gabriel, Miguel A; Olza Fernández, Ibone; Malalana Martínez, Ana M; González Armengod, Carmen; Costarelli, Valeria; Millán Santos, Isabel; Fernández-Cañadas Morillo, Aurora; Pérez Riveiro, Pilar; López Sánchez, Francisco; García Murillo, Lourdes

发表日期：2015

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The output of the tRNA modification pathways controlled by the Escherichia coli MnmEG and MnmC enzymes depends on the growth conditions and the tRNA species

大肠杆菌 MnmEG 和 MnmC 酶控制的 tRNA 修饰途径的输出取决于生长条件和 tRNA 种类

期刊:Nucleic Acids Research

影响因子:16.6

doi:10.1093/nar/gkt1228

Ismaïl Moukadiri, M-José Garzón, Glenn R Björk, M-Eugenia Armengod

表观遗传

发表日期：2014

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Thrombotic microangiopathy associated with lenvatinib therapy

与乐伐替尼治疗相关的血栓性微血管病

The MELAS mutation m.3243A>G alters the expression of mitochondrial tRNA fragments

MELAS 突变 m.3243A>G 改变了线粒体 tRNA 片段的表达

The MELAS mutation m.3243A>G promotes reactivation of fetal cardiac genes and an epithelial-mesenchymal transition-like program via dysregulation of miRNAs

MELAS 突变 m.3243A>G 通过 miRNA 失调促进胎儿心脏基因的重新激活和上皮-间质转化样程序

Defects in the mitochondrial-tRNA modification enzymes MTO1 and GTPBP3 promote different metabolic reprogramming through a HIF-PPARγ-UCP2-AMPK axis

线粒体 tRNA 修饰酶 MTO1 和 GTPBP3 的缺陷通过 HIF-PPARγ-UCP2-AMPK 轴促进不同的代谢重编程

microRNA-mediated differential expression of TRMU, GTPBP3 and MTO1 in cell models of mitochondrial-DNA diseases

microRNA 介导的线粒体 DNA 疾病细胞模型中 TRMU、GTPBP3 和 MTO1 的差异表达

Mutations in the Caenorhabditis elegans orthologs of human genes required for mitochondrial tRNA modification cause similar electron transport chain defects but different nuclear responses

线粒体 tRNA 修饰所需的人类基因的秀丽隐杆线虫直系同源物的突变会导致类似的电子传递链缺陷，但会导致不同的核反应

The ROS-sensitive microRNA-9/9* controls the expression of mitochondrial tRNA-modifying enzymes and is involved in the molecular mechanism of MELAS syndrome

ROS 敏感的 microRNA-9/9* 控制线粒体 tRNA 修饰酶的表达，并参与 MELAS 综合征的分子机制

Defective Expression of the Mitochondrial-tRNA Modifying Enzyme GTPBP3 Triggers AMPK-Mediated Adaptive Responses Involving Complex I Assembly Factors, Uncoupling Protein 2, and the Mitochondrial Pyruvate Carrier

线粒体 tRNA 修饰酶 GTPBP3 表达缺陷会触发 AMPK 介导的适应性反应，涉及复合物 I 组装因子、解偶联蛋白 2 和线粒体丙酮酸载体

Intrapartum synthetic oxytocin reduce the expression of primitive reflexes associated with breastfeeding

产时合成催产素会降低与母乳喂养相关的原始反射的表达。

The output of the tRNA modification pathways controlled by the Escherichia coli MnmEG and MnmC enzymes depends on the growth conditions and the tRNA species

大肠杆菌 MnmEG 和 MnmC 酶控制的 tRNA 修饰途径的输出取决于生长条件和 tRNA 种类