日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Chromosomal Instability and Telomere Attrition in Systemic Sclerosis: A Historical Perspective

系统性硬化症中的染色体不稳定性和端粒缩短:历史视角

Artlett, Carol M

Soluble Receptor for Glycation End-products Concentration Increases Following the Treatment of Severe Diabetic Ketoacidosis.

严重糖尿病酮症酸中毒治疗后,糖化终产物可溶性受体浓度增加

Hoffman William H, Ishikawa Takaki, Blum James, Tani Naoto, Ikeda Tomoya, Artlett Carol M

Mir-155 is overexpressed in systemic sclerosis fibroblasts and is required for NLRP3 inflammasome-mediated collagen synthesis during fibrosis

Mir-155在系统性硬化症成纤维细胞中过度表达,并且是纤维化过程中NLRP3炎症小体介导的胶原合成所必需的。

Artlett, Carol M; Sassi-Gaha, Sihem; Hope, Jennifer L; Feghali-Bostwick, Carol A; Katsikis, Peter D

Chimeric cells of maternal origin do not appear to be pathogenic in the juvenile idiopathic inflammatory myopathies or muscular dystrophy

母源嵌合细胞似乎不会引起青少年特发性炎症性肌病或肌营养不良症。

Artlett, Carol M; Sassi-Gaha, Sihem; Ramos, Ronald C; Miller, Frederick W; Rider, Lisa G

Tight Skin 2 Mice Exhibit Delayed Wound Healing Caused by Increased Elastic Fibers in Fibrotic Skin

紧致皮肤2小鼠表现出伤口愈合延迟,这是由于纤维化皮肤中弹性纤维增加所致。

Long, Kristen B; Burgwin, Chelsea M; Huneke, Richard; Artlett, Carol M; Blankenhorn, Elizabeth P

Targeting the unfolded protein response, XBP1, and the NLRP3 inflammasome in fibrosis and cancer

靶向未折叠蛋白反应、XBP1 和 NLRP3 炎症小体在纤维化和癌症中的作用

Overley-Adamson, Beth; Artlett, Carol M; Stephens, Connie; Sassi-Gaha, Sihem; Weis, Ransome D; Thacker, James D

NLRP3 inflammasome is a target for development of broad-spectrum anti-infective drugs

NLRP3炎症小体是开发广谱抗感染药物的靶点

Thacker, James D; Balin, Brian J; Appelt, Denah M; Sassi-Gaha, Sihem; Purohit, Mitali; Rest, Richard F; Artlett, Carol M

Methodology for Detecting Trace Amounts of Microchimeric DNA from Peripheral Murine White Blood Cells by Real-Time PCR

利用实时PCR检测小鼠外周血白细胞中痕量微嵌合DNA的方法

Artlett, Carol M; Dito, C Gennaro; Christner, Paul J