Nicotine inhibits the production of proinflammatory mediators in human monocytes by suppression of I-kappaB phosphorylation and nuclear factor-kappaB transcriptional activity through nicotinic acetylcholine receptor alpha7
尼古丁通过抑制I-κB磷酸化和核因子-κB转录活性，进而抑制人单核细胞中促炎介质的产生，这一过程是通过尼古丁乙酰胆碱受体α7亚基实现的。
期刊:Clinical and Experimental Immunology
影响因子:3.8
doi:10.1111/j.1365-2249.2006.03169.x
Yoshikawa, H; Kurokawa, M; Ozaki, N; Nara, K; Atou, K; Takada, E; Kamochi, H; Suzuki, N
