Avner Ellis D相关文献与解析，生知库 | 链接生命科学的每一步

New approaches to the autosomal recessive polycystic kidney disease patient with dual kidney-liver complications

针对伴有肾肝双重并发症的常染色体隐性多囊肾病患者，提出了新的治疗方法。

期刊:Pediatric Transplantation

影响因子:1.4

doi:10.1111/petr.12076

Telega, Grzegorz; Cronin, David; Avner, Ellis D

发表日期：2013

文献求助收藏

Transport, cilia, and PKD: must we in (cyst) on interrelationships? Focus on "Increased Na+/H+ exchanger activity on the apical surface of a cilium-deficient cortical collecting duct principal cell model of polycystic kidney disease"

运输、纤毛和多囊肾：我们必须关注它们之间的相互关系吗？重点关注“多囊肾病纤毛缺陷皮质集合管主细胞模型顶端表面Na+/H+交换活性增强”。

期刊:American Journal of Physiology-Cell Physiology

影响因子:4.7

doi:10.1152/ajpcell.00070.2012

Avner, Ellis D; McDonough, Alicia A; Sweeney, William E Jr

细胞生物学

发表日期：2012

文献求助收藏

Role of genetic modifiers in an orthologous rat model of ARPKD

遗传修饰因子在ARPKD同源大鼠模型中的作用

期刊:Physiological Genomics

影响因子:2.5

doi:10.1152/physiolgenomics.00187.2011

O'Meara, Caitlin C; Hoffman, Matthew; Sweeney, William E Jr; Tsaih, Shirng-Wern; Xiao, Bing; Jacob, Howard J; Avner, Ellis D; Moreno, Carol

Rat

发表日期：2012

文献求助收藏

Loss of activator of G-protein signaling 3 impairs renal tubular regeneration following acute kidney injury in rodents.

G蛋白信号激活因子3的缺失会损害啮齿动物急性肾损伤后的肾小管再生

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.10-169797

Regner Kevin R, Nozu Kandai, Lanier Stephen M, Blumer Joe B, Avner Ellis D, Sweeney William E Jr, Park Frank

20-HETE activates the Raf/MEK/ERK pathway in renal epithelial cells through an EGFR- and c-Src-dependent mechanism.

20-HETE 通过 EGFR 和 c-Src 依赖性机制激活肾上皮细胞中的 Raf/MEK/ERK 通路

期刊:American Journal of Physiology-Renal Physiology

影响因子:3.4

doi:10.1152/ajprenal.00146.2009

Akbulut Talha, Regner Kevin R, Roman Richard J, Avner Ellis D, Falck John R, Park Frank

20-HETE mediates proliferation of renal epithelial cells in polycystic kidney disease

20-HETE介导多囊肾病中肾上皮细胞的增殖

期刊:Journal of the American Society of Nephrology

影响因子:9.4

doi:10.1681/ASN.2007070771

Park, Frank; Sweeney, William E; Jia, Guangfu; Roman, Richard J; Avner, Ellis D

Emerging Therapies for Childhood Polycystic Kidney Disease

儿童多囊肾病的新兴疗法

Smac-mimetic-induced epithelial cell death reduces the growth of renal cysts

Smac模拟物诱导的上皮细胞死亡可减少肾囊肿的生长

New approaches to the autosomal recessive polycystic kidney disease patient with dual kidney-liver complications

针对伴有肾肝双重并发症的常染色体隐性多囊肾病患者，提出了新的治疗方法。

Transport, cilia, and PKD: must we in (cyst) on interrelationships? Focus on "Increased Na+/H+ exchanger activity on the apical surface of a cilium-deficient cortical collecting duct principal cell model of polycystic kidney disease"

运输、纤毛和多囊肾：我们必须关注它们之间的相互关系吗？重点关注“多囊肾病纤毛缺陷皮质集合管主细胞模型顶端表面Na+/H+交换活性增强”。

Role of genetic modifiers in an orthologous rat model of ARPKD

遗传修饰因子在ARPKD同源大鼠模型中的作用

Loss of activator of G-protein signaling 3 impairs renal tubular regeneration following acute kidney injury in rodents.

G蛋白信号激活因子3的缺失会损害啮齿动物急性肾损伤后的肾小管再生

20-HETE activates the Raf/MEK/ERK pathway in renal epithelial cells through an EGFR- and c-Src-dependent mechanism.

20-HETE 通过 EGFR 和 c-Src 依赖性机制激活肾上皮细胞中的 Raf/MEK/ERK 通路

20-HETE mediates proliferation of renal epithelial cells in polycystic kidney disease

20-HETE介导多囊肾病中肾上皮细胞的增殖