Calsequestrin 2 deletion shortens the refractoriness of Ca²⁺ release and reduces rate-dependent Ca²⁺-alternans in intact mouse hearts

钙结合蛋白2缺失缩短了完整小鼠心脏中Ca²⁺释放的不应期，并降低了速率依赖性Ca²⁺交替现象