日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

The Oxysterol Synthesising Enzyme CH25H Contributes to the Development of Intestinal Fibrosis

氧固醇合成酶 CH25H 促进肠道纤维化的发展

T Raselli, A Wyss, M N Gonzalez Alvarado, B Weder, C Mamie, M R Spalinger, W T Van Haaften, G Dijkstra, A W Sailer, P H Imenez Silva, C A Wagner, V Tosevski, Sebastian Leibl, M Scharl, G Rogler, M Hausmann, B Misselwitz

BCL-2 levels do not predict azathioprine treatment response in inflammatory bowel disease, but inhibition induces lymphocyte apoptosis and ameliorates colitis in mice

BCL-2 水平不能预测硫唑嘌呤治疗炎症性肠病的疗效,但抑制 BCL-2 可诱导淋巴细胞凋亡并改善小鼠结肠炎。

B Weder ,M Mozaffari ,L Biedermann ,C Mamie ,A Moncsek ,L Wang ,S H Clarke ,G Rogler ,B L McRae ,C L Graff ,P A Ruiz ,M Hausmann

A critical review of the current knowledge regarding the biological impact of nanocellulose

对纳米纤维素生物学影响的最新研究进展进行批判性综述

Endes, C; Camarero-Espinosa, S; Mueller, S; Foster, E J; Petri-Fink, A; Rothen-Rutishauser, B; Weder, C; Clift, M J D

Combined Genetic and Genealogic Studies Uncover a Large BAP1 Cancer Syndrome Kindred Tracing Back Nine Generations to a Common Ancestor from the 1700s

结合遗传学和家谱学研究,发现一个庞大的BAP1癌症综合征家族,其家族成员可追溯至18世纪的共同祖先,共九代。

Carbone, Michele; Flores, Erin G; Emi, Mitsuru; Johnson, Todd A; Tsunoda, Tatsuhiko; Behner, Dusty; Hoffman, Harriet; Hesdorffer, Mary; Nasu, Masaki; Napolitano, Andrea; Powers, Amy; Minaai, Michael; Baumann, Francine; Bryant-Greenwood, Peter; Lauk, Olivia; Kirschner, Michaela B; Weder, Walter; Opitz, Isabelle; Pass, Harvey I; Gaudino, Giovanni; Pastorino, Sandra; Yang, Haining

A polymorphic 3'UTR element in ATP1B1 regulates alternative polyadenylation and is associated with blood pressure

ATP1B1 中的多态性 3'UTR 元件可调节替代性多聚腺苷酸化并与血压相关

Megana K Prasad, Kavita Bhalla, Zhen Hua Pan, Jeffrey R O'Connell, Alan B Weder, Aravinda Chakravarti, Bin Tian, Yen-Pei C Chang

Human leukocyte antigen G up-regulation in lung cancer associates with high-grade histology, human leukocyte antigen class I loss and interleukin-10 production

肺癌中人类白细胞抗原G(HLA-G)表达上调与高级别组织学特征、HLA-I类分子丢失和白细胞介素-10(IL-10)产生相关。

Urosevic, M; Kurrer, M O; Kamarashev, J; Mueller, B; Weder, W; Burg, G; Stahel, R A; Dummer, R; Trojan, A