Babaev Vladimir R相关文献与解析，生知库

Macrophage IKKα Deficiency Suppresses Akt Phosphorylation, Reduces Cell Survival, and Decreases Early Atherosclerosis

巨噬细胞 IKKα 缺乏抑制 Akt 磷酸化，降低细胞存活率，并减少早期动脉粥样硬化

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.115.306931

Babaev, Vladimir R; Ding, Lei; Zhang, Youmin; May, James M; Lin, P Charles; Fazio, Sergio; Linton, MacRae F

Jnk1 Deficiency in Hematopoietic Cells Suppresses Macrophage Apoptosis and Increases Atherosclerosis in Low-Density Lipoprotein Receptor Null Mice

造血细胞中Jnk1缺陷抑制巨噬细胞凋亡并增加低密度脂蛋白受体敲除小鼠的动脉粥样硬化

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.116.307580

Babaev, Vladimir R; Yeung, Michele; Erbay, Ebru; Ding, Lei; Zhang, Youmin; May, James M; Fazio, Sergio; Hotamisligil, Gökhan S; Linton, MacRae F

Macrophage Apoptosis and Efferocytosis in the Pathogenesis of Atherosclerosis

巨噬细胞凋亡和胞吞作用在动脉粥样硬化发病机制中的作用

期刊:Circulation Journal

影响因子:3.7

doi:10.1253/circj.CJ-16-0924

Linton, MacRae F; Babaev, Vladimir R; Huang, Jiansheng; Linton, Edward F; Tao, Huan; Yancey, Patricia G

Macrophage SR-BI mediates efferocytosis via Src/PI3K/Rac1 signaling and reduces atherosclerotic lesion necrosis.

巨噬细胞 SR-BI 通过 Src/PI3K/Rac1 信号传导介导胞吞作用，并减少动脉粥样硬化病变坏死

期刊:Journal of Lipid Research

影响因子:4.1

doi:10.1194/jlr.M056689

Tao Huan, Yancey Patricia G, Babaev Vladimir R, Blakemore John L, Zhang Youmin, Ding Lei, Fazio Sergio, Linton MacRae F

细胞生物学

发表日期：2015

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Identification of small proline-rich repeat protein 3 as a novel atheroprotective factor that promotes adaptive Akt signaling in vascular smooth muscle cells

鉴定出富含脯氨酸重复序列的小蛋白3是一种新型抗动脉粥样硬化因子，它能促进血管平滑肌细胞中适应性Akt信号通路的激活。

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.114.303644

Segedy, Amanda K; Pyle, Amy L; Li, Bin; Zhang, Youmin; Babaev, Vladimir R; Jat, Parmjit; Fazio, Sergio; Atkinson, James B; Linton, MacRae F; Young, Pampee P

Combined vitamin C and vitamin E deficiency worsens early atherosclerosis in apolipoprotein E-deficient mice

维生素C和维生素E联合缺乏会加重载脂蛋白E缺陷小鼠的早期动脉粥样硬化。

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.110.209502

Babaev, Vladimir R; Li, Liying; Shah, Sanket; Fazio, Sergio; Linton, MacRae F; May, James M

Reducing endoplasmic reticulum stress through a macrophage lipid chaperone alleviates atherosclerosis

通过巨噬细胞脂质伴侣减轻内质网应激可缓解动脉粥样硬化

期刊:Nature Medicine

影响因子:50

doi:10.1038/nm.2067

Erbay, Ebru; Babaev, Vladimir R; Mayers, Jared R; Makowski, Liza; Charles, Khanichi N; Snitow, Melinda E; Fazio, Sergio; Wiest, Michelle M; Watkins, Steven M; Linton, Macrae F; Hotamisligil, Gökhan S

Macrophage EP4 deficiency increases apoptosis and suppresses early atherosclerosis

巨噬细胞EP4缺陷会增加细胞凋亡并抑制早期动脉粥样硬化

期刊:Cell Metabolism

影响因子:30.9

doi:10.1016/j.cmet.2008.09.005

Babaev, Vladimir R; Chew, Joshua D; Ding, Lei; Davis, Sarah; Breyer, Matthew D; Breyer, Richard M; Oates, John A; Fazio, Sergio; Linton, Macrae F

Imaging mass spectrometry reveals heterogeneity of proliferation and metabolism in atherosclerosis

成像质谱法揭示动脉粥样硬化中增殖和代谢的异质性

Akt Signaling in Macrophage Polarization, Survival, and Atherosclerosis

Akt信号通路在巨噬细胞极化、存活和动脉粥样硬化中的作用

Macrophage IKKα Deficiency Suppresses Akt Phosphorylation, Reduces Cell Survival, and Decreases Early Atherosclerosis

巨噬细胞 IKKα 缺乏抑制 Akt 磷酸化，降低细胞存活率，并减少早期动脉粥样硬化

Jnk1 Deficiency in Hematopoietic Cells Suppresses Macrophage Apoptosis and Increases Atherosclerosis in Low-Density Lipoprotein Receptor Null Mice

造血细胞中Jnk1缺陷抑制巨噬细胞凋亡并增加低密度脂蛋白受体敲除小鼠的动脉粥样硬化

Macrophage Apoptosis and Efferocytosis in the Pathogenesis of Atherosclerosis

巨噬细胞凋亡和胞吞作用在动脉粥样硬化发病机制中的作用

Macrophage SR-BI mediates efferocytosis via Src/PI3K/Rac1 signaling and reduces atherosclerotic lesion necrosis.

巨噬细胞 SR-BI 通过 Src/PI3K/Rac1 信号传导介导胞吞作用，并减少动脉粥样硬化病变坏死

Identification of small proline-rich repeat protein 3 as a novel atheroprotective factor that promotes adaptive Akt signaling in vascular smooth muscle cells

鉴定出富含脯氨酸重复序列的小蛋白3是一种新型抗动脉粥样硬化因子，它能促进血管平滑肌细胞中适应性Akt信号通路的激活。

Combined vitamin C and vitamin E deficiency worsens early atherosclerosis in apolipoprotein E-deficient mice

维生素C和维生素E联合缺乏会加重载脂蛋白E缺陷小鼠的早期动脉粥样硬化。

Reducing endoplasmic reticulum stress through a macrophage lipid chaperone alleviates atherosclerosis

通过巨噬细胞脂质伴侣减轻内质网应激可缓解动脉粥样硬化

Macrophage EP4 deficiency increases apoptosis and suppresses early atherosclerosis

巨噬细胞EP4缺陷会增加细胞凋亡并抑制早期动脉粥样硬化