Babbette相关文献与解析，生知库 | 链接生命科学的每一步

Thomas Herrock, Owen; Campbell, Nathan; Deer, Evangeline; Amaral, Lorena M; Whitney, Darby; Morris, Rachael; Wallace, Kedra; Warren Turner, Ty; Cleveland, E Hawthorne; Belk, Sheila; Booz, George W; Cornelius, Denise C; LaMarca, Babbette

心血管

B细胞

免疫/内分泌

Immune Cells in Preeclampsia

先兆子痫中的免疫细胞

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms27010074

Campbell, Nathan; Robbins, Marcus; Nembaware, Hellen; Deer, Evangeline; Cornelius, Denise; LaMarca, Babbette

The Role of Mitochondrial Dysfunction and Oxidative Stress in Women's Reproductive Disorders: Implications for Polycystic Ovary Syndrome and Preeclampsia

线粒体功能障碍和氧化应激在女性生殖系统疾病中的作用：对多囊卵巢综合征和先兆子痫的启示

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms26136439

Deer, Evangeline; LaMarca, Babbette; Reckelhoff, Jane F; Shawky, Noha M; Edwards, Kristin

AT1-AA induced hypertension during pregnancy contributes to developmental programming of hypertension in female offspring

妊娠期AT1-AA诱导的高血压会导致雌性后代高血压的发育编程。

期刊:Journal of Reproductive Immunology

影响因子:

doi:10.1016/j.jri.2025.104802

Campbell, Nathan; Lemon, James P; Helmich, Melissa; Solise, Dylan; Demesa, Alexandra; Goolsby, Ella; LaMarca, Evan; Deer, Evangeline; Cornelius, Denise C; Turner, Ty; Amaral, Lorena M; Zheng, Baoying; LaMarca, Babbette

AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring Health

AT1-AA在胎盘缺血后于子代体内产生，B细胞耗竭可降低其水平，但不会影响子代的整体健康。

期刊:Journal of the American Heart Association

影响因子:5.3

doi:10.1161/JAHA.123.031417

Campbell, Nathan; Deer, Evangeline; Solise, Dylan; Cornelius, Denise C; Turner, Ty; Amaral, Lorena M; Herrock, Owen; Jordan, Ariel; Shukla, Shivani; Ibrahim, Tarek; LaMarca, Babbette

The role of T cell stimulated agonistic autoantibodies to the angiotensin II type I receptor (AT1-AA) in mediating multiorgan dysfunction in IL-17 induced hypertension during pregnancy

T细胞刺激产生的针对血管紧张素II 1型受体（AT1-AA）的激动性自身抗体在IL-17诱导的妊娠期高血压介导的多器官功能障碍中的作用

期刊:American Journal of Reproductive Immunology

影响因子:2.4

doi:10.1111/aji.13843

Hogg, James P 3rd; Campbell, Nathan; Deer, Evangeline; Fitzgerald, Sarah; Cornelius, Denise; Hoang, Ngoc; Turner, Ty; Amaral, Lorena; Lemon, James P; Ibrahim, Tarek; LaMarca, Babbette

The role of immune cells and mediators in preeclampsia

免疫细胞和介质在先兆子痫中的作用

期刊:Nature Reviews Nephrology

影响因子:39.8

doi:10.1038/s41581-022-00670-0

Deer, Evangeline; Herrock, Owen; Campbell, Nathan; Cornelius, Denise; Fitzgerald, Sarah; Amaral, Lorena M; LaMarca, Babbette

B2 cells contribute to hypertension and natural killer cell activation possibly via AT1-AA in response to placental ischemia

B2 细胞可能通过 AT1-AA 促进高血压和自然杀伤细胞活化，以应对胎盘缺血

期刊:American Journal of Physiology-Renal Physiology

影响因子:3.7

doi:10.1152/ajprenal.00190.2022

Owen T Herrock, Evangeline Deer, Lorena M Amaral, Nathan Campbell, James Lemon, Nicole Ingram, Denise C Cornelius, Ty W Turner, Sarah Fitzgerald, Tarek Ibrahim, Ralf Dechend, Gerd Wallukat, Babbette LaMarca

Inhibition of angiotensin II type 1 receptor agonistic autoantibodies by direct binding does not impact reduced uterine perfusion pressure offspring birthweight and blood pressure at adulthood.

通过直接结合抑制血管紧张素 II 1 型受体激动剂自身抗体，不会影响子宫灌注压降低、后代出生体重和成年血压

期刊:American Journal of Obstetrics & Gynecology Mfm

影响因子:3.1

doi:10.1016/j.ajogmf.2023.100945

Solise Dylan, Campbell Nathan, Ashraf Usman, Herrock Owen, Crudup Breland, Mallette Jordan, Willis Alex, Rawls Adam Z, Turner Ty, Cockrell Kathy, Zheng Baoying, Deer Evangeline, Amaral Lorena, Alexander Barbara T, Lamarca Babbette

RUPP Th17s cause hypertension and mitochondrial dysfunction in the kidney and placenta during pregnancy

RUPP Th17s 会导致妊娠期高血压以及肾脏和胎盘线粒体功能障碍。

期刊:Pregnancy Hypertension-An International Journal of Womens Cardiovascular Health

影响因子:2.9

doi:10.1016/j.preghy.2023.04.002

Fitzgerald, Sarah; Deer, Evangeline; Hogg, James; Cornelius, Denise C; Turner, Ty; Amaral, Lorena M; Hoang, Ngoc; Edwards, Kristin; Herrock, Owen; Campbell, Nathan; Ibrahim, Tarek; LaMarca, Babbette

Preeclamptic Placental CD19+ B Cells Are Causal to Hypertension During Pregnancy

子痫前期胎盘CD19+ B细胞是妊娠期高血压的病因

Immune Cells in Preeclampsia

先兆子痫中的免疫细胞

The Role of Mitochondrial Dysfunction and Oxidative Stress in Women's Reproductive Disorders: Implications for Polycystic Ovary Syndrome and Preeclampsia

线粒体功能障碍和氧化应激在女性生殖系统疾病中的作用：对多囊卵巢综合征和先兆子痫的启示

AT1-AA induced hypertension during pregnancy contributes to developmental programming of hypertension in female offspring

妊娠期AT1-AA诱导的高血压会导致雌性后代高血压的发育编程。

AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring Health

AT1-AA在胎盘缺血后于子代体内产生，B细胞耗竭可降低其水平，但不会影响子代的整体健康。

The role of T cell stimulated agonistic autoantibodies to the angiotensin II type I receptor (AT1-AA) in mediating multiorgan dysfunction in IL-17 induced hypertension during pregnancy

T细胞刺激产生的针对血管紧张素II 1型受体（AT1-AA）的激动性自身抗体在IL-17诱导的妊娠期高血压介导的多器官功能障碍中的作用

The role of immune cells and mediators in preeclampsia

免疫细胞和介质在先兆子痫中的作用

B2 cells contribute to hypertension and natural killer cell activation possibly via AT1-AA in response to placental ischemia

B2 细胞可能通过 AT1-AA 促进高血压和自然杀伤细胞活化，以应对胎盘缺血

Inhibition of angiotensin II type 1 receptor agonistic autoantibodies by direct binding does not impact reduced uterine perfusion pressure offspring birthweight and blood pressure at adulthood.

通过直接结合抑制血管紧张素 II 1 型受体激动剂自身抗体，不会影响子宫灌注压降低、后代出生体重和成年血压

RUPP Th17s cause hypertension and mitochondrial dysfunction in the kidney and placenta during pregnancy

RUPP Th17s 会导致妊娠期高血压以及肾脏和胎盘线粒体功能障碍。