Banks Lawrence相关文献与解析，生知库 | 链接生命科学的每一步

Genetic Diversity of Selected High-Risk HPV Types Prevalent in Africa and Not Covered by Current Vaccines: A Pooled Sequence Data Analysis

非洲流行且现有疫苗未覆盖的特定高危型HPV的遗传多样性：一项汇总序列数据分析

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms262211056

Nyide, Babalwa; Thomas, Miranda; Banks, Lawrence; Mkhize, Pamela P; Matume, Nontokozo D

发表日期：2025

文献求助收藏

Nucleic acid nanobiosystems for cancer theranostics: an overview of emerging trends and challenges

用于癌症诊疗一体化的核酸纳米生物系统：新兴趋势与挑战概述

期刊:Nanomedicine

影响因子:3.9

doi:10.1080/17435889.2025.2501919

Rebolledo, Laura P; Andrade, Luciana N S; Bajgelman, Marcio C; Banks, Lawrence; Breakefield, Xandra O; Dobrovolskaia, Marina A; Dokholyan, Nikolay V; Kimura, Edna T; Villa, Luisa; Zerbini, Luiz F; Zucolotto, Valtencir; Afonin, Kirill A; Strauss, Bryan E; Chammas, Roger; de Freitas Saito, Renata

The retinoblastoma protein contributes to maintaining the stability of HPV E7 in cervical cancer cells.

视网膜母细胞瘤蛋白有助于维持宫颈癌细胞中 HPV E7 的稳定性

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/jvi.02203-24

Gbala Ifeoluwa, Kavcic Nezka, Banks Lawrence

细胞生物学

发表日期：2025

文献求助收藏

HPV16 E7 modulates the cell surface expression of MET and CD109 via the AP2 complex

HPV16 E7 通过 AP2 复合物调节 MET 和 CD109 的细胞表面表达。

期刊:Tumour Virus Research

影响因子:8.1

doi:10.1016/j.tvr.2024.200279

Trejo-Cerro, Oscar; Basukala, Om; Myers, Michael P; Banks, Lawrence

HPV-18 E6 enhances the interaction between EMILIN2 and SNX27 to promote WNT signaling

HPV-18 E6 增强 EMILIN2 和 SNX27 之间的相互作用，从而促进 WNT 信号传导。

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/jvi.00735-24

Broniarczyk, Justyna; Trejo-Cerro, Oscar; Massimi, Paola; Kavčič, Nežka; Myers, Michael P; Banks, Lawrence

信号转导

发表日期：2024

文献求助收藏

Molecular dissection of the E6 PBM identifies essential residues regulating Chk1 phosphorylation and subsequent 14-3-3 recognition

E6 PBM 的分子解析鉴定出调控 Chk1 磷酸化及其后续 14-3-3 识别的关键残基。

期刊:Tumour Virus Research

影响因子:8.1

doi:10.1016/j.tvr.2023.200257

Vats, Arushi; Thatte, Jayashree V; Banks, Lawrence

Correction: Clinical validation of full HR-HPV genotyping HPV Selfy assay according to the international guidelines for HPV test requirements for cervical cancer screening on clinician-collected and self-collected samples

更正：根据国际HPV检测指南，对临床医生采集和患者自采样本进行HPV Selfy全基因分型检测，以验证其在宫颈癌筛查中的临床有效性。

期刊:Journal of Translational Medicine

影响因子:7.5

doi:10.1186/s12967-023-03882-5

Avian, Alice; Clemente, Nicolò; Mauro, Elisabetta; Isidoro, Erica; Di Napoli, Michela; Dudine, Sandra; Del Fabro, Anna; Morini, Stefano; Perin, Tiziana; Giudici, Fabiola; Cammisuli, Tamara; Foschi, Nicola; Mocenigo, Marco; Montrone, Michele; Modena, Chiara; Polenghi, Martina; Puzzi, Luca; Tomaic, Vjekoslav; Valenti, Giulio; Sola, Riccardo; Zanolla, Shivani; Vogrig, Enea; Riva, Elisabetta; Angeletti, Silvia; Ciccozzi, Massimo; Castriciano, Santina; Pachetti, Maria; Petti, Matteo; Centonze, Sandro; Gerin, Daniela; Banks, Lawrence; Marini, Bruna; Canzonieri, Vincenzo; Sopracordevole, Francesco; Zanconati, Fabrizio; Ippodrino, Rudy

Clinical validation of full HR-HPV genotyping HPV Selfy assay according to the international guidelines for HPV test requirements for cervical cancer screening on clinician-collected and self-collected samples

根据国际HPV检测指南，对临床医生采集和患者自采样本进行HPV Selfy全基因分型检测，以验证其在宫颈癌筛查中的应用。

期刊:Journal of Translational Medicine

影响因子:7.5

doi:10.1186/s12967-022-03383-x

Avian, Alice; Clemente, Nicolò; Mauro, Elisabetta; Isidoro, Erica; Di Napoli, Michela; Dudine, Sandra; Del Fabro, Anna; Morini, Stefano; Perin, Tiziana; Giudici, Fabiola; Cammisuli, Tamara; Foschi, Nicola; Mocenigo, Marco; Montrone, Michele; Modena, Chiara; Polenghi, Martina; Puzzi, Luca; Tomaic, Vjekoslav; Valenti, Giulio; Sola, Riccardo; Zanolla, Shivani; Vogrig, Enea; Riva, Elisabetta; Angeletti, Silvia; Ciccozzi, Massimo; Castriciano, Santina; Pachetti, Maria; Petti, Matteo; Centonze, Sandro; Gerin, Daniela; Banks, Lawrence; Marini, Bruna; Canzonieri, Vincenzo; Sopracordevole, Francesco; Zanconati, Fabrizio; Ippodrino, Rudy

HPV-18E6 Inhibits Interactions between TANC2 and SNX27 in a PBM-Dependent Manner and Promotes Increased Cell Proliferation

HPV-18E6 以 PBM 依赖的方式抑制 TANC2 和 SNX27 之间的相互作用，并促进细胞增殖。

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/jvi.01365-22

Broniarczyk, Justyna Karolina; Massimi, Paola; Trejo-Cerro, Oscar; Myers, Michael P; Banks, Lawrence

Editorial: Tumour Virus Research on Virus Host Interactions and Cell Transformation

社论：肿瘤病毒研究——病毒宿主相互作用和细胞转化

Genetic Diversity of Selected High-Risk HPV Types Prevalent in Africa and Not Covered by Current Vaccines: A Pooled Sequence Data Analysis

非洲流行且现有疫苗未覆盖的特定高危型HPV的遗传多样性：一项汇总序列数据分析

Nucleic acid nanobiosystems for cancer theranostics: an overview of emerging trends and challenges

用于癌症诊疗一体化的核酸纳米生物系统：新兴趋势与挑战概述

The retinoblastoma protein contributes to maintaining the stability of HPV E7 in cervical cancer cells.

视网膜母细胞瘤蛋白有助于维持宫颈癌细胞中 HPV E7 的稳定性

HPV16 E7 modulates the cell surface expression of MET and CD109 via the AP2 complex

HPV16 E7 通过 AP2 复合物调节 MET 和 CD109 的细胞表面表达。

HPV-18 E6 enhances the interaction between EMILIN2 and SNX27 to promote WNT signaling

HPV-18 E6 增强 EMILIN2 和 SNX27 之间的相互作用，从而促进 WNT 信号传导。

Molecular dissection of the E6 PBM identifies essential residues regulating Chk1 phosphorylation and subsequent 14-3-3 recognition

E6 PBM 的分子解析鉴定出调控 Chk1 磷酸化及其后续 14-3-3 识别的关键残基。

Correction: Clinical validation of full HR-HPV genotyping HPV Selfy assay according to the international guidelines for HPV test requirements for cervical cancer screening on clinician-collected and self-collected samples

更正：根据国际HPV检测指南，对临床医生采集和患者自采样本进行HPV Selfy全基因分型检测，以验证其在宫颈癌筛查中的临床有效性。

Clinical validation of full HR-HPV genotyping HPV Selfy assay according to the international guidelines for HPV test requirements for cervical cancer screening on clinician-collected and self-collected samples

根据国际HPV检测指南，对临床医生采集和患者自采样本进行HPV Selfy全基因分型检测，以验证其在宫颈癌筛查中的应用。

HPV-18E6 Inhibits Interactions between TANC2 and SNX27 in a PBM-Dependent Manner and Promotes Increased Cell Proliferation

HPV-18E6 以 PBM 依赖的方式抑制 TANC2 和 SNX27 之间的相互作用，并促进细胞增殖。