日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

The CaMKII D135N mutation blocks kinase activity and reduces GluN2B binding

CaMKII D135N 突变会阻断激酶活性并降低 GluN2B 的结合。

Larsen, Matthew E; Barker, C Madison; Vargas, Raul Satoshi; Coultrap, Steven J; Bayer, K Ulrich

CaMKII monomers are sufficient for GluN2B binding, co-condensation, and synaptic potentiation.

CaMKII 单体足以与 GluN2B 结合、共凝聚和突触增强

Brown Carolyn Nicole, Barker C Madison, Miller Carley N, Aoto Jason, Coultrap Steven J, Bayer K Ulrich

LTP expression mediated by autonomous activity of GluN2B-bound CaMKII.

LTP表达由GluN2B结合的CaMKII的自主活性介导

Rumian Nicole L, Barker C Madison, Larsen Matthew E, Tullis Jonathan E, Freund Ronald K, Taslimi Amir, Coultrap Steven J, Tucker Chandra L, Dell'Acqua Mark L, Bayer K Ulrich