Bartsch Udo相关文献与解析，生知库 | 链接生命科学的每一步

Survival and Axonal Regeneration of Retinal Ganglion Cells in a Mouse Optic Nerve Crush Model After a Cell-Based Intravitreal Co-Administration of Ciliary Neurotrophic Factor and Glial Cell Line-Derived Neurotrophic Factor at Different Post-Lesion Time Points.

在小鼠视神经挤压模型中，不同损伤后时间点进行基于细胞的玻璃体内联合注射睫状神经营养因子和胶质细胞系衍生神经营养因子后，视网膜神经节细胞的存活和轴突再生情况

期刊:Cells

影响因子:5.2

doi:10.3390/cells14090643

Hu Yue, Grodzki Lynn Michelle, Bartsch Udo

Experimental Therapeutic Approaches for the Treatment of Retinal Pathology in Neuronal Ceroid Lipofuscinoses

神经元蜡样脂褐质沉积症视网膜病变的实验性治疗方法

期刊:Frontiers in Neurology

影响因子:2.8

doi:10.3389/fneur.2022.866983

Bartsch, Udo; Storch, Stephan

Cell-Based Neuroprotection of Retinal Ganglion Cells in Animal Models of Optic Neuropathies

在视神经病变动物模型中，基于细胞的视网膜神经节细胞神经保护作用

期刊:Biology-Basel

影响因子:3.5

doi:10.3390/biology10111181

Hu, Yue; Grodzki, Lynn Michelle; Bartsch, Susanne; Bartsch, Udo

细胞生物学

发表日期：2021

文献求助收藏

Intravitreal Co-Administration of GDNF and CNTF Confers Synergistic and Long-Lasting Protection against Injury-Induced Cell Death of Retinal Ganglion Cells in Mice

玻璃体内联合注射 GDNF 和 CNTF 可对小鼠视网膜神经节细胞损伤诱导的细胞死亡产生协同和持久的保护作用

期刊:Cells

影响因子:5.1

doi:10.3390/cells9092082

Simon Dulz, Mahmoud Bassal, Kai Flachsbarth, Kristoffer Riecken, Boris Fehse, Stefanie Schlichting, Susanne Bartsch, Udo Bartsch

Current and Emerging Treatment Strategies for Neuronal Ceroid Lipofuscinoses

神经元蜡样脂褐质沉积症的当前和新兴治疗策略

期刊:CNS Drugs

影响因子:7.4

doi:10.1007/s40263-019-00620-8

Kohlschütter, Alfried; Schulz, Angela; Bartsch, Udo; Storch, Stephan

神经科学

发表日期：2019

文献求助收藏

Genetically modified neural stem cells for a local and sustained delivery of neuroprotective factors to the dystrophic mouse retina

利用基因改造的神经干细胞，将神经保护因子局部且持续地输送到营养不良的小鼠视网膜。

期刊:Stem Cells Translational Medicine

影响因子:4.9

doi:10.5966/sctm.2013-0013

Jung, Gila; Sun, Jing; Petrowitz, Bettina; Riecken, Kristoffer; Kruszewski, Katharina; Jankowiak, Wanda; Kunst, Frank; Skevas, Christos; Richard, Gisbert; Fehse, Boris; Bartsch, Udo

Lack of a-disintegrin-and-metalloproteinase ADAM10 leads to intracellular accumulation and loss of shedding of the cellular prion protein in vivo

体内缺乏α-解整合素和金属蛋白酶ADAM10会导致细胞内朊病毒蛋白的积累和脱落丧失。

期刊:Molecular Neurodegeneration

影响因子:17.5

doi:10.1186/1750-1326-6-36

Altmeppen, Hermann C; Prox, Johannes; Puig, Berta; Kluth, Mark A; Bernreuther, Christian; Thurm, Dana; Jorissen, Ellen; Petrowitz, Bettina; Bartsch, Udo; De Strooper, Bart; Saftig, Paul; Glatzel, Markus

Terminal differentiation of myelin-forming oligodendrocytes depends on the transcription factor Sox10

髓鞘形成少突胶质细胞的终末分化依赖于转录因子Sox10

期刊:Genes & Development

影响因子:7.7

doi:10.1101/gad.215802

Stolt, C Claus; Rehberg, Stephan; Ader, Marius; Lommes, Petra; Riethmacher, Dieter; Schachner, Melitta; Bartsch, Udo; Wegner, Michael

Atp1b2(Atp1b1) Knock-In Mice Exhibit a Cone-Rod Dystrophy-Like Phenotype.

Atp1b2(Atp1b1)敲入小鼠表现出类似视锥细胞-视杆细胞营养不良的表型

Survival and Axonal Regeneration of Retinal Ganglion Cells in a Mouse Optic Nerve Crush Model After a Cell-Based Intravitreal Co-Administration of Ciliary Neurotrophic Factor and Glial Cell Line-Derived Neurotrophic Factor at Different Post-Lesion Time Points.

在小鼠视神经挤压模型中，不同损伤后时间点进行基于细胞的玻璃体内联合注射睫状神经营养因子和胶质细胞系衍生神经营养因子后，视网膜神经节细胞的存活和轴突再生情况

Experimental Therapeutic Approaches for the Treatment of Retinal Pathology in Neuronal Ceroid Lipofuscinoses

神经元蜡样脂褐质沉积症视网膜病变的实验性治疗方法

Cell-Based Neuroprotection of Retinal Ganglion Cells in Animal Models of Optic Neuropathies

在视神经病变动物模型中，基于细胞的视网膜神经节细胞神经保护作用

Intravitreal Co-Administration of GDNF and CNTF Confers Synergistic and Long-Lasting Protection against Injury-Induced Cell Death of Retinal Ganglion Cells in Mice

玻璃体内联合注射 GDNF 和 CNTF 可对小鼠视网膜神经节细胞损伤诱导的细胞死亡产生协同和持久的保护作用

Current and Emerging Treatment Strategies for Neuronal Ceroid Lipofuscinoses

神经元蜡样脂褐质沉积症的当前和新兴治疗策略

Genetically modified neural stem cells for a local and sustained delivery of neuroprotective factors to the dystrophic mouse retina

利用基因改造的神经干细胞，将神经保护因子局部且持续地输送到营养不良的小鼠视网膜。

Lack of a-disintegrin-and-metalloproteinase ADAM10 leads to intracellular accumulation and loss of shedding of the cellular prion protein in vivo

体内缺乏α-解整合素和金属蛋白酶ADAM10会导致细胞内朊病毒蛋白的积累和脱落丧失。

Terminal differentiation of myelin-forming oligodendrocytes depends on the transcription factor Sox10

髓鞘形成少突胶质细胞的终末分化依赖于转录因子Sox10