Basler Michael相关文献与解析，生知库 | 链接生命科学的每一步

Immunoproteasome subunits are novel signatures for predicting efficacy of immunotherapy in muscle invasive bladder cancer

免疫蛋白酶体亚基是预测肌层浸润性膀胱癌免疫疗法疗效的新型标志物

Wang, XinJian; You, Hang; Zhang, Teng; Li, Yuan; Chen, XinYu; Basler, Michael; Jiang, QingMing; Chen, Han; Liu, Nan; Yuan, Fang; Li, Jun

肿瘤

Immunoproteasome inhibition triggers protein stress and apoptosis in cells of B cell lineage without impairing vaccination-induced antibody responses

免疫蛋白酶体抑制可诱导B细胞谱系细胞发生蛋白质应激和凋亡，但不会影响疫苗诱导的抗体反应。

期刊:Cell Death Discovery

影响因子:7

doi:10.1038/s41420-025-02818-w

Mink, Dennis; Oliveri, Franziska; Otto, Julia; Kutsi, Nazlim; Gonzalez Siebold, Carolina; Muchamuel, Tony; Li, Jun; Basler, Michael

Chemical and Biological Mechanisms Relevant to the Rescue of MG-132-Treated Neurons by Cysteine.

半胱氨酸拯救 MG-132 处理的神经元的化学和生物学机制

期刊:Antioxidants

影响因子:6.6

doi:10.3390/antiox14020128

Ãckert Anna-Katharina, Suciu Ilinca, Land Anja, Spreng Anna-Sophie, Welte Hannah, Herzog Doreen, Basler Michael, Leist Marcel

神经科学

发表日期：2025

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The ubiquitin-like modifier FAT10 is not essential for MHC-I antigen presentation

类泛素修饰因子FAT10并非MHC-I抗原呈递所必需。

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2025.1636951

Pach, Natalie; Ochs, Sarah; Cao, Jinjing; Ottlinger, Julia; Aichem, Annette; Basler, Michael

FAT1

发表日期：2025

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Immunoproteasome Inhibition Impairs Differentiation but Not Survival of T Helper 17 Cells

免疫蛋白酶体抑制会损害T辅助细胞17的分化，但不会影响其存活。

期刊:Cells

影响因子:5.2

doi:10.3390/cells14100689

Oliveri, Franziska; Mink, Dennis; Muchamuel, Tony; Basler, Michael

scTEA-db: a comprehensive database of novel terminal exon isoforms identified from human single cell transcriptomes

scTEA-db：一个包含从人类单细胞转录组中鉴定出的新型末端外显子异构体的综合数据库

期刊:Nucleic Acids Research

影响因子:13.1

doi:10.1093/nar/gkad878

Barquin, Miguel; Kouzel, Ian U; Ehrmann, Beat; Basler, Michael; Gruber, Andreas J

Cellular stress increases DRIP production and MHC Class I antigen presentation.

细胞应激会增加 DRIP 的产生和 MHC I 类抗原的呈递

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2024.1445338

Pach Natalie, Basler Michael

细胞生物学

发表日期：2024

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Phosphorylated FAT10 Is More Efficiently Conjugated to Substrates, Does Not Bind to NUB1L, and Does Not Alter Degradation by the Proteasome

磷酸化的FAT10能更有效地与底物结合，不与NUB1L结合，也不改变蛋白酶体的降解作用。

期刊:Biomedicines

影响因子:3.9

doi:10.3390/biomedicines12122795

Cao, Jinjing; Aichem, Annette; Basler, Michael; Alvarez Salinas, Gerardo Omar; Schmidtke, Gunter

Suppression of prostate cancer and amelioration of the immunosuppressive tumor microenvironment through selective immunoproteasome inhibition

通过选择性免疫蛋白酶体抑制抑制前列腺癌并改善免疫抑制性肿瘤微环境

期刊:Oncoimmunology

影响因子:6.3

doi:10.1080/2162402X.2022.2156091

Koerner, Julia; Horvath, Dennis; Oliveri, Franziska; Li, Jun; Basler, Michael

Intact lymph node homing and CD8(+) T-cell priming abilities of Sprouty2-deficient dendritic cells.

Sprouty2 缺陷型树突状细胞的淋巴结归巢和 CD8(+) T 细胞启动能力完整。

Immunoproteasome subunits are novel signatures for predicting efficacy of immunotherapy in muscle invasive bladder cancer

免疫蛋白酶体亚基是预测肌层浸润性膀胱癌免疫疗法疗效的新型标志物

Immunoproteasome inhibition triggers protein stress and apoptosis in cells of B cell lineage without impairing vaccination-induced antibody responses

免疫蛋白酶体抑制可诱导B细胞谱系细胞发生蛋白质应激和凋亡，但不会影响疫苗诱导的抗体反应。

Chemical and Biological Mechanisms Relevant to the Rescue of MG-132-Treated Neurons by Cysteine.

半胱氨酸拯救 MG-132 处理的神经元的化学和生物学机制

The ubiquitin-like modifier FAT10 is not essential for MHC-I antigen presentation

类泛素修饰因子FAT10并非MHC-I抗原呈递所必需。

Immunoproteasome Inhibition Impairs Differentiation but Not Survival of T Helper 17 Cells

免疫蛋白酶体抑制会损害T辅助细胞17的分化，但不会影响其存活。

scTEA-db: a comprehensive database of novel terminal exon isoforms identified from human single cell transcriptomes

scTEA-db：一个包含从人类单细胞转录组中鉴定出的新型末端外显子异构体的综合数据库

Cellular stress increases DRIP production and MHC Class I antigen presentation.

细胞应激会增加 DRIP 的产生和 MHC I 类抗原的呈递

Phosphorylated FAT10 Is More Efficiently Conjugated to Substrates, Does Not Bind to NUB1L, and Does Not Alter Degradation by the Proteasome

磷酸化的FAT10能更有效地与底物结合，不与NUB1L结合，也不改变蛋白酶体的降解作用。

Suppression of prostate cancer and amelioration of the immunosuppressive tumor microenvironment through selective immunoproteasome inhibition

通过选择性免疫蛋白酶体抑制抑制前列腺癌并改善免疫抑制性肿瘤微环境