日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Transcriptomic Signatures of End-Stage Human Dilated Cardiomyopathy Hearts with and without Left Ventricular Assist Device Support

终末期扩张型心肌病患者心脏(有/无左心室辅助装置支持)的转录组特征

Parikh, Mihir; Shah, Saumya; Basu, Ratnadeep; Famulski, Konrad S; Kim, Daniel; Mullen, John C; Halloran, Philip F; Oudit, Gavin Y

PI3Kα Pathway Inhibition With Doxorubicin Treatment Results in Distinct Biventricular Atrophy and Remodeling With Right Ventricular Dysfunction

多柔比星治疗抑制PI3Kα通路可导致明显的双心室萎缩和重塑,并伴有右心室功能障碍

McLean, Brent A; Patel, Vaibhav B; Zhabyeyev, Pavel; Chen, Xueyi; Basu, Ratnadeep; Wang, Faqi; Shah, Saumya; Vanhaesebroeck, Bart; Oudit, Gavin Y

Advanced iron-overload cardiomyopathy in a genetic murine model is rescued by resveratrol therapy

在基因小鼠模型中,晚期铁过载性心肌病可通过白藜芦醇疗法得到挽救。

Das, Subhash K; Zhabyeyev, Pavel; Basu, Ratnadeep; Patel, Vaibhav B; Dyck, Jason R B; Kassiri, Zamaneh; Oudit, Gavin Y

TIMP3 deficiency exacerbates iron overload-mediated cardiomyopathy and liver disease

TIMP3 缺乏会加剧铁过载介导的心肌病和肝病。

Zhabyeyev, Pavel; Das, Subhash K; Basu, Ratnadeep; Shen, Mengcheng; Patel, Vaibhav B; Kassiri, Zamaneh; Oudit, Gavin Y

Females Are Protected From Iron-Overload Cardiomyopathy Independent of Iron Metabolism: Key Role of Oxidative Stress

女性免受铁过载性心肌病的影响,且这种保护作用与铁代谢无关:氧化应激发挥关键作用

Das, Subhash K; Patel, Vaibhav B; Basu, Ratnadeep; Wang, Wang; DesAulniers, Jessica; Kassiri, Zamaneh; Oudit, Gavin Y

ACE2 Deficiency Worsens Epicardial Adipose Tissue Inflammation and Cardiac Dysfunction in Response to Diet-Induced Obesity

ACE2 缺乏会加剧饮食诱导肥胖引起的心外膜脂肪组织炎症和心脏功能障碍

Patel, Vaibhav B; Mori, Jun; McLean, Brent A; Basu, Ratnadeep; Das, Subhash K; Ramprasath, Tharmarajan; Parajuli, Nirmal; Penninger, Josef M; Grant, Maria B; Lopaschuk, Gary D; Oudit, Gavin Y

Loss of angiotensin-converting enzyme-2 exacerbates diabetic cardiovascular complications and leads to systolic and vascular dysfunction: a critical role of the angiotensin II/AT1 receptor axis

血管紧张素转换酶-2的缺失会加剧糖尿病心血管并发症,并导致收缩功能障碍和血管功能障碍:血管紧张素II/AT1受体轴发挥着关键作用。

Patel, Vaibhav B; Bodiga, Sreedhar; Basu, Ratnadeep; Das, Subhash K; Wang, Wang; Wang, Zuocheng; Lo, Jennifer; Grant, Maria B; Zhong, JiuChang; Kassiri, Zamaneh; Oudit, Gavin Y

Early activation of matrix metalloproteinases underlies the exacerbated systolic and diastolic dysfunction in mice lacking TIMP3 following myocardial infarction.

心肌梗死后,缺乏 TIMP3 的小鼠出现收缩和舒张功能障碍加剧,其原因是基质金属蛋白酶的早期激活

Kandalam Vijay, Basu Ratnadeep, Abraham Thomas, Wang Xiuhua, Awad Ahmed, Wang Wei, Lopaschuk Gary D, Maeda Nobuyo, Oudit Gavin Y, Kassiri Zamaneh