Bauer Stephen M相关文献与解析，生知库 | 链接生命科学的每一步

Developmental exposure to bisphenol A modulates innate but not adaptive immune responses to influenza A virus infection

发育过程中接触双酚A会调节先天性免疫反应，但不会影响对甲型流感病毒感染的适应性免疫反应。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0038448

Roy, Anirban; Bauer, Stephen M; Lawrence, B Paige

Activation of pulmonary dendritic cells and Th2-type inflammatory responses on instillation of engineered, environmental diesel emission source or ambient air pollutant particles in vivo

体内滴注人工合成的环境柴油排放源或环境空气污染物颗粒后，肺树突状细胞被激活，并引发Th2型炎症反应。

期刊:Journal of Innate Immunity

影响因子:3

doi:10.1159/000321725

Bezemer, Gillina F G; Bauer, Stephen M; Oberdörster, Günter; Breysse, Patrick N; Pieters, Raymond H H; Georas, Steve N; Williams, Marc A

Disruption of the transcription factor Nrf2 promotes pro-oxidative dendritic cells that stimulate Th2-like immunoresponsiveness upon activation by ambient particulate matter.

转录因子 Nrf2 的破坏会促进促氧化树突状细胞的产生，这些细胞在被环境颗粒物激活后会刺激 Th2 样免疫反应

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.181.7.4545

Williams Marc A, Rangasamy Tirumalai, Bauer Stephen M, Killedar Smruti, Karp Matthew, Kensler Thomas W, Yamamoto Masayuki, Breysse Patrick, Biswal Shyam, Georas Steve N

Diesel-enriched particulate matter functionally activates human dendritic cells

富含柴油的颗粒物可激活人类树突状细胞的功能。

期刊:American Journal of Respiratory Cell and Molecular Biology

影响因子:5.3

doi:10.1165/rcmb.2007-0199OC

Porter, Michael; Karp, Matthew; Killedar, Smruti; Bauer, Stephen M; Guo, Jia; Williams, D'Ann; Breysse, Patrick; Georas, Steve N; Williams, Marc A

The effects of maternal exposure to bisphenol A on allergic lung inflammation into adulthood

母体接触双酚A对成年期过敏性肺部炎症的影响

Developmental exposure to bisphenol A modulates innate but not adaptive immune responses to influenza A virus infection

发育过程中接触双酚A会调节先天性免疫反应，但不会影响对甲型流感病毒感染的适应性免疫反应。

Activation of pulmonary dendritic cells and Th2-type inflammatory responses on instillation of engineered, environmental diesel emission source or ambient air pollutant particles in vivo

体内滴注人工合成的环境柴油排放源或环境空气污染物颗粒后，肺树突状细胞被激活，并引发Th2型炎症反应。

Disruption of the transcription factor Nrf2 promotes pro-oxidative dendritic cells that stimulate Th2-like immunoresponsiveness upon activation by ambient particulate matter.

转录因子 Nrf2 的破坏会促进促氧化树突状细胞的产生，这些细胞在被环境颗粒物激活后会刺激 Th2 样免疫反应

Diesel-enriched particulate matter functionally activates human dendritic cells

富含柴油的颗粒物可激活人类树突状细胞的功能。