Benter Ibrahim F相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Targeting the epidermal growth factor receptor (EGFR/ErbB) for the potential treatment of renal pathologies

靶向表皮生长因子受体（EGFR/ErbB）治疗肾脏疾病

期刊:Frontiers in Pharmacology

影响因子:4.8

doi:10.3389/fphar.2024.1394997

Tawengi, Mohamed; Al-Dali, Yazan; Tawengi, Abdelaziz; Benter, Ibrahim F; Akhtar, Saghir

发表日期：2024

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Mitigating Cardiotoxicity of Dendrimers: Angiotensin-(1-7) via Its Mas Receptor Ameliorates PAMAM-Induced Cardiac Dysfunction in the Isolated Mammalian Heart

减轻树状聚合物的心脏毒性：血管紧张素-(1-7)通过其Mas受体改善PAMAM诱导的离体哺乳动物心脏功能障碍

期刊:Pharmaceutics

影响因子:5.5

doi:10.3390/pharmaceutics14122673

Akhtar, Saghir; Babiker, Fawzi; Akhtar, Usman A; Benter, Ibrahim F

发表日期：2022

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The Role of Epidermal Growth Factor Receptor Family of Receptor Tyrosine Kinases in Mediating Diabetes-Induced Cardiovascular Complications

表皮生长因子受体家族受体酪氨酸激酶在介导糖尿病诱发的心血管并发症中的作用

期刊:Frontiers in Pharmacology

影响因子:4.8

doi:10.3389/fphar.2021.701390

Shraim, Bara A; Moursi, Moaz O; Benter, Ibrahim F; Habib, Abdella M; Akhtar, Saghir

发表日期：2021

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Nanotoxicology of Dendrimers in the Mammalian Heart: ex vivo and in vivo Administration of G6 PAMAM Nanoparticles Impairs Recovery of Cardiac Function Following Ischemia-Reperfusion Injury

树状聚合物在哺乳动物心脏中的纳米毒理学研究：离体和体内实验表明，G6 PAMAM纳米颗粒的给药会损害缺血再灌注损伤后心脏功能的恢复。

期刊:International Journal of Nanomedicine

影响因子:6.5

doi:10.2147/IJN.S255202

Babiker, Fawzi; Benter, Ibrahim F; Akhtar, Saghir

发表日期：2020

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Ang-(1-7)/ MAS1 receptor axis inhibits allergic airway inflammation via blockade of Src-mediated EGFR transactivation in a murine model of asthma

在哮喘小鼠模型中，Ang-(1-7)/MAS1受体轴通过阻断Src介导的EGFR反式激活来抑制过敏性气道炎症。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0224163

El-Hashim, Ahmed Z; Khajah, Maitham A; Babyson, Rhema S; Renno, Waleed M; Ezeamuzie, Charles I; Benter, Ibrahim F; Akhtar, Saghir

发表日期：2019

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Src-dependent EGFR transactivation regulates lung inflammation via downstream signaling involving ERK1/2, PI3Kδ/Akt and NFκB induction in a murine asthma model

在小鼠哮喘模型中，Src依赖的EGFR反式激活通过下游信号通路（包括ERK1/2、PI3Kδ/Akt和NFκB的诱导）调节肺部炎症。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-017-09349-0

El-Hashim, Ahmed Z; Khajah, Maitham A; Renno, Waleed M; Babyson, Rhema S; Uddin, Mohib; Benter, Ibrahim F; Ezeamuzie, Charles; Akhtar, Saghir

发表日期：2017

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Cationic Polyamidoamine Dendrimers as Modulators of EGFR Signaling In Vitro and In Vivo.

阳离子聚酰胺胺树状聚合物作为体外和体内 EGFR 信号传导的调节剂

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0132215

Akhtar Saghir, Al-Zaid Bashayer, El-Hashim Ahmed Z, Chandrasekhar Bindu, Attur Sreeja, Yousif Mariam H M, Benter Ibrahim F

发表日期：2015

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Chronic treatment with Ang-(1-7) reverses abnormal reactivity in the corpus cavernosum and normalizes diabetes-induced changes in the protein levels of ACE, ACE2, ROCK1, ROCK2 and omega-hydroxylase in a rat model of type 1 diabetes

在1型糖尿病大鼠模型中，长期使用Ang-(1-7)治疗可逆转阴茎海绵体的异常反应性，并使糖尿病引起的ACE、ACE2、ROCK1、ROCK2和ω-羟化酶蛋白水平的变化恢复正常。

期刊:Journal of Diabetes Research

影响因子:3.4

doi:10.1155/2014/142154

Yousif, Mariam H M; Makki, Batoul; El-Hashim, Ahmed Z; Akhtar, Saghir; Benter, Ibrahim F

发表日期：2014

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RU28318, an aldosterone antagonist, in combination with an ACE inhibitor and angiotensin receptor blocker attenuates cardiac dysfunction in diabetes

醛固酮拮抗剂RU28318与血管紧张素转换酶抑制剂和血管紧张素受体阻滞剂联合使用，可减轻糖尿病患者的心脏功能障碍。

期刊:Journal of Diabetes Research

影响因子:3.4

doi:10.1155/2013/427693

Benter, Ibrahim F; Babiker, Fawzi; Al-Rashdan, Ibrahim; Yousif, Mariam; Akhtar, Saghir

发表日期：2013

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Angiotensin-(1-7) via the mas receptor alleviates the diabetes-induced decrease in GFAP and GAP-43 immunoreactivity with concomitant reduction in the COX-2 in hippocampal formation: an immunohistochemical study.

血管紧张素-(1-7)通过mas受体减轻糖尿病引起的GFAP和GAP-43免疫反应性降低，同时降低海马结构中的COX-2：一项免疫组织化学研究

期刊:Cellular and Molecular Neurobiology

影响因子:4.8

doi:10.1007/s10571-012-9858-7

Renno Waleed M, Al-Banaw Anwar G, George Preethi, Abu-Ghefreh Asmaa A, Akhtar Saghir, Benter Ibrahim F

发表日期：2012

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