日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Dynamics and mechanisms of CNS myelination

中枢神经系统髓鞘形成的动力学和机制

Bercury, Kathryn K; Macklin, Wendy B

Olig1 Acetylation and Nuclear Export Mediate Oligodendrocyte Development

Olig1乙酰化和核输出介导少突胶质细胞发育

Jinxiang Dai, Kathryn K Bercury, Weilin Jin, Wendy B Macklin

Olig1 function is required for oligodendrocyte differentiation in the mouse brain

Olig1 功能是小鼠大脑少突胶质细胞分化所必需的

Jinxiang Dai, Kathryn K Bercury, Jared T Ahrendsen, Wendy B Macklin

Interaction of mTOR and Erk1/2 signaling to regulate oligodendrocyte differentiation

mTOR 与 Erk1/2 信号相互作用调控少突胶质细胞分化

JinXiang Dai, Kathryn K Bercury, Wendy B Macklin

Conditional ablation of raptor or rictor has differential impact on oligodendrocyte differentiation and CNS myelination

条件性消融猛禽或rictor对少突胶质细胞分化和中枢神经系统髓鞘形成有不同的影响

Kathryn K Bercury, JinXiang Dai, Hilary H Sachs, Jared T Ahrendsen, Teresa L Wood, Wendy B Macklin

Mammalian target of rapamycin promotes oligodendrocyte differentiation, initiation and extent of CNS myelination

雷帕霉素的哺乳动物靶点促进少突胶质细胞的分化、中枢神经系统髓鞘的启动和程度

Stacey E Wahl, Lauren E McLane, Kathryn K Bercury, Wendy B Macklin, Teresa L Wood

A new model of cuprizone-mediated demyelination/remyelination

铜宗介导的脱髓鞘/髓鞘再生新模型

Hilary H Sachs, Kathryn K Bercury, Daniela C Popescu, S Priya Narayanan, Wendy B Macklin

mTOR: a link from the extracellular milieu to transcriptional regulation of oligodendrocyte development

mTOR:连接细胞外环境与少突胶质细胞发育转录调控的桥梁

Wood, Teresa L; Bercury, Kathryn K; Cifelli, Stacey E; Mursch, Lauren E; Min, Jungsoo; Dai, Jinxiang; Macklin, Wendy B

Systemic delivery of a glucosylceramide synthase inhibitor reduces CNS substrates and increases lifespan in a mouse model of type 2 Gaucher disease

在2型戈谢病小鼠模型中,全身性给予葡糖基神经酰胺合成酶抑制剂可减少中枢神经系统底物并延长寿命。

Cabrera-Salazar, Mario A; Deriso, Matthew; Bercury, Scott D; Li, Lingyun; Lydon, John T; Weber, William; Pande, Nilesh; Cromwell, Mandy A; Copeland, Diane; Leonard, John; Cheng, Seng H; Scheule, Ronald K

Iminosugar-based inhibitors of glucosylceramide synthase increase brain glycosphingolipids and survival in a mouse model of Sandhoff disease

基于亚氨基糖的葡糖基神经酰胺合成酶抑制剂可增加桑德霍夫病小鼠模型的脑糖鞘脂含量并提高其存活率。

Ashe, Karen M; Bangari, Dinesh; Li, Lingyun; Cabrera-Salazar, Mario A; Bercury, Scott D; Nietupski, Jennifer B; Cooper, Christopher G F; Aerts, Johannes M F G; Lee, Edward R; Copeland, Diane P; Cheng, Seng H; Scheule, Ronald K; Marshall, John