Bettelli Estelle相关文献与解析，生知库 | 链接生命科学的每一步

Correction: Cutting Edge: Integrin α(4) Is Required for Regulatory B Cell Control of Experimental Autoimmune Encephalomyelitis

更正：前沿：整合素α(4)是调节性B细胞控制实验性自身免疫性脑脊髓炎所必需的

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1700283

Glatigny, Simon; Wagner, Catriona A; Bettelli, Estelle

Cutting Edge: Integrin α4 Is Required for Regulatory B Cell Control of Experimental Autoimmune Encephalomyelitis

前沿：整合素α4是调节性B细胞控制实验性自身免疫性脑脊髓炎所必需的

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1502614

Glatigny, Simon; Wagner, Catriona A; Bettelli, Estelle

Integrin alpha L controls the homing of regulatory T cells during CNS autoimmunity in the absence of integrin alpha 4

在缺乏整合素α4的情况下，整合素αL控制中枢神经系统自身免疫过程中调节性T细胞的归巢。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/srep07834

Glatigny, Simon; Duhen, Rebekka; Arbelaez, Carlos; Kumari, Swarnima; Bettelli, Estelle

Cutting edge: the pathogenicity of IFN-Î³-producing Th17 cells is independent of T-bet.

前沿发现：产生 IFN-γ 的 Th17 细胞的致病性与 T-bet 无关

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1203172

Duhen Rebekka, Glatigny Simon, Arbelaez Carlos A, Blair Tiffany C, Oukka Mohamed, Bettelli Estelle

Proinflammatory T helper type 17 cells are effective B-cell helpers

促炎性T辅助细胞17型是有效的B细胞辅助细胞

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.1009234107

Mitsdoerffer, Meike; Lee, Youjin; Jäger, Anneli; Kim, Hye-Jung; Korn, Thomas; Kolls, Jay K; Cantor, Harvey; Bettelli, Estelle; Kuchroo, Vijay K

Myelin-specific regulatory T cells accumulate in the CNS but fail to control autoimmune inflammation

髓鞘特异性调节性T细胞在中枢神经系统中积聚，但无法控制自身免疫性炎症。

期刊:Nature Medicine

影响因子:50

doi:10.1038/nm1564

Korn, Thomas; Reddy, Jayagopala; Gao, Wenda; Bettelli, Estelle; Awasthi, Amit; Petersen, Troels R; Bäckström, B Thomas; Sobel, Raymond A; Wucherpfennig, Kai W; Strom, Terry B; Oukka, Mohamed; Kuchroo, Vijay K

IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells

IL-21启动一条替代途径诱导促炎性T(H)17细胞

期刊:Nature

影响因子:48.5

doi:10.1038/nature05970

Korn, Thomas; Bettelli, Estelle; Gao, Wenda; Awasthi, Amit; Jäger, Anneli; Strom, Terry B; Oukka, Mohamed; Kuchroo, Vijay K

Experimental Autoimmune Encephalomyelitis (EAE) as Animal Models of Multiple Sclerosis (MS)

实验性自身免疫性脑脊髓炎（EAE）作为多发性硬化症（MS）的动物模型

Regulation of lymphocyte trafficking in central nervous system autoimmunity

中枢神经系统自身免疫中淋巴细胞迁移的调控

S1P(1) deletion differentially affects TH17 and Regulatory T cells

S1P(1)缺失对TH17细胞和调节性T细胞的影响不同

Correction: Cutting Edge: Integrin α(4) Is Required for Regulatory B Cell Control of Experimental Autoimmune Encephalomyelitis

更正：前沿：整合素α(4)是调节性B细胞控制实验性自身免疫性脑脊髓炎所必需的

Cutting Edge: Integrin α4 Is Required for Regulatory B Cell Control of Experimental Autoimmune Encephalomyelitis

前沿：整合素α4是调节性B细胞控制实验性自身免疫性脑脊髓炎所必需的

Integrin alpha L controls the homing of regulatory T cells during CNS autoimmunity in the absence of integrin alpha 4

在缺乏整合素α4的情况下，整合素αL控制中枢神经系统自身免疫过程中调节性T细胞的归巢。

Cutting edge: the pathogenicity of IFN-Î³-producing Th17 cells is independent of T-bet.

前沿发现：产生 IFN-γ 的 Th17 细胞的致病性与 T-bet 无关

Proinflammatory T helper type 17 cells are effective B-cell helpers

促炎性T辅助细胞17型是有效的B细胞辅助细胞

Myelin-specific regulatory T cells accumulate in the CNS but fail to control autoimmune inflammation

髓鞘特异性调节性T细胞在中枢神经系统中积聚，但无法控制自身免疫性炎症。

IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells

IL-21启动一条替代途径诱导促炎性T(H)17细胞