日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Toll-like Receptor 9 in Preclinical Models of Idiopathic Pulmonary Fibrosis

特发性肺纤维化临床前模型中的 Toll 样受体 9

Verma, Priyanka; Hamill, Timothy; Bhattacharyya, Swati

Author Correction: Fibroblast A20 governs fibrosis susceptibility and its repression by DREAM promotes fibrosis in multiple organs

作者更正:成纤维细胞A20控制纤维化易感性,而DREAM对其的抑制会促进多个器官的纤维化。

Wang, Wenxia; Bale, Swarna; Wei, Jun; Yalavarthi, Bharath; Bhattacharyya, Dibyendu; Yan, Jing Jing; Abdala-Valencia, Hiam; Xu, Dan; Sun, Hanshi; Marangoni, Roberta G; Herzog, Erica; Berdnikovs, Sergejs; Miller, Stephen D; Sawalha, Amr H; Tsou, Pei-Suen; Awaji, Kentaro; Yamashita, Takashi; Sato, Shinichi; Asano, Yoshihide; Tiruppathi, Chinnaswamy; Yeldandi, Anjana; Schock, Bettina C; Bhattacharyya, Swati; Varga, John

Extracellular Matrix-Derived Damage-Associated Molecular Patterns (DAMP): Implications in Systemic Sclerosis and Fibrosis

细胞外基质衍生的损伤相关分子模式(DAMP):在系统性硬化症和纤维化中的意义

Bale, Swarna; Verma, Priyanka; Varga, John; Bhattacharyya, Swati

Tenascin-C in fibrosis in multiple organs: Translational implications

腱生蛋白-C在多器官纤维化中的作用:转化意义

Bhattacharyya, Swati; Midwood, Kim S; Varga, John

Targeting CD38-dependent NAD(+) metabolism to mitigate multiple organ fibrosis

靶向 CD38 依赖性 NAD(+) 代谢以减轻多器官纤维化

Shi, Bo; Wang, Wenxia; Korman, Benjamin; Kai, Li; Wang, Qianqian; Wei, Jun; Bale, Swarna; Marangoni, Roberta Goncalves; Bhattacharyya, Swati; Miller, Stephen; Xu, Dan; Akbarpour, Mahzad; Cheresh, Paul; Proccissi, Daniele; Gursel, Demirkan; Espindola-Netto, Jair Machado; Chini, Claudia C S; de Oliveira, Guilherme C; Gudjonsson, Johann E; Chini, Eduardo N; Varga, John

Matrix protein tenascin-C expands and reversibly blocks maturation of murine eosinophil progenitors

基质蛋白腱生蛋白-C可扩增并可逆性地阻断小鼠嗜酸性粒细胞祖细胞的成熟。

Doan, Ton C; Jeong, Brian M; Coden, Mackenzie E; Loffredo, Lucas F; Bhattacharyya, Swati; Chiarella, Sergio E; Varga, John; Abdala-Valencia, Hiam; Berdnikovs, Sergejs

Pharmacological Inhibition of Toll-Like Receptor-4 Signaling by TAK242 Prevents and Induces Regression of Experimental Organ Fibrosis

TAK242通过药理学抑制Toll样受体4信号通路,可预防和诱导实验性器官纤维化的消退。

Bhattacharyya, Swati; Wang, Wenxia; Tamaki, Zenshiro; Shi, Bo; Yeldandi, Anjana; Tsukimi, Yasuhiro; Yamasaki, Masashi; Varga, John

Fibronectin EDA forms the chronic fibrotic scar after contusive spinal cord injury

纤维连接蛋白EDA在脊髓挫伤后形成慢性纤维化瘢痕

Cooper, John G; Jeong, Su Ji; McGuire, Tammy L; Sharma, Sripadh; Wang, Wenxia; Bhattacharyya, Swati; Varga, John; Kessler, John A

Endogenous ligands of TLR4 promote unresolving tissue fibrosis: Implications for systemic sclerosis and its targeted therapy

TLR4的内源性配体促进难消退性组织纤维化:对系统性硬化症及其靶向治疗的启示

Bhattacharyya, Swati; Varga, John

Toll-Like Receptor-4 Signaling Drives Persistent Fibroblast Activation and Prevents Fibrosis Resolution in Scleroderma

Toll样受体4信号通路驱动成纤维细胞持续活化并阻止硬皮病纤维化消退

Bhattacharyya, Swati; Midwood, Kim S; Yin, Hang; Varga, John