日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Role of interleukin-6 in hemopoietic and non-hemopoietic synergy mediating TLR4-triggered late murine ileus and endotoxic shock.

白细胞介素-6在造血和非造血协同作用介导TLR4触发的小鼠晚期肠梗阻和内毒素休克中的作用

Buchholz B M, Chanthaphavong R S, Billiar T R, Bauer A J

TLR4/MyD88-induced CD11b+Gr-1 int F4/80+ non-migratory myeloid cells suppress Th2 effector function in the lung

TLR4/MyD88诱导的CD11b+Gr-1 int F4/80+非迁移性髓系细胞抑制肺部Th2效应功能

Arora, M; Poe, S L; Oriss, T B; Krishnamoorthy, N; Yarlagadda, M; Wenzel, S E; Billiar, T R; Ray, A; Ray, P

The acute inflammatory response in trauma / hemorrhage and traumatic brain injury: current state and emerging prospects

创伤/出血和创伤性脑损伤中的急性炎症反应:现状与展望

Namas, R; Ghuma, A; Hermus, L; Zamora, R; Okonkwo, D O; Billiar, T R; Vodovotz, Y

Molecular mechanisms in the early phase of hemorrhagic shock

出血性休克早期阶段的分子机制

Hierholzer, C; Billiar, T R

Pan-Caspase Inhibitor Prevents Anti-CD3 Mediated T-Lymphocyte Proliferation

泛半胱天冬酶抑制剂可阻止抗CD3介导的T淋巴细胞增殖

Mahidhara, Raja S; Hoffman, R; Simmons, R L; Billiar, T R

Essential role of induced nitric oxide in the initiation of the inflammatory response after hemorrhagic shock

诱导型一氧化氮在出血性休克后炎症反应启动中的关键作用

Hierholzer, C; Harbrecht, B; Menezes, J M; Kane, J; MacMicking, J; Nathan, C F; Peitzman, A B; Billiar, T R; Tweardy, D J

Role of nitric oxide in inflammation and tissue injury during endotoxemia and hemorrhagic shock

一氧化氮在内毒素血症和出血性休克引起的炎症和组织损伤中的作用

Shah, N S; Billiar, T R

Roles of nitric oxide in surgical infection and sepsis

一氧化氮在外科感染和脓毒症中的作用

Johnson, M L; Billiar, T R

Inducible nitric oxide synthase suppresses the development of allograft arteriosclerosis

诱导型一氧化氮合酶抑制同种异体移植动脉硬化的发展

Shears, L L; Kawaharada, N; Tzeng, E; Billiar, T R; Watkins, S C; Kovesdi, I; Lizonova, A; Pham, S M

Transcriptional regulation of human inducible nitric oxide synthase (NOS2) gene by cytokines: initial analysis of the human NOS2 promoter

细胞因子对人诱导型一氧化氮合酶 (NOS2) 基因转录调控:人 NOS2 启动子的初步分析

de Vera, M E; Shapiro, R A; Nussler, A K; Mudgett, J S; Simmons, R L; Morris, S M Jr; Billiar, T R; Geller, D A