日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

CD8+ T cells drive myofibroblast activation and contraction via JAK/STAT3 and TGFβ signaling

CD8+ T 细胞通过 JAK/STAT3 和 TGFβ 信号通路驱动肌成纤维细胞活化和收缩

Theodoros Ioannis Papadimitriou ,Anne van Essen ,Merel Willemse ,Daphne Dorst ,Elly Vitters ,Birgitte Walgreen ,Hans J P M Koenen ,Peter van der Kraan ,Marije Koenders ,Rogier M Thurlings ,Arjan van Caam

Local inhibition of TGF-β1 signaling improves Th17/Treg balance but not joint pathology during experimental arthritis

局部抑制 TGF-β1 信号可改善实验性关节炎期间 Th17/Treg 平衡,但不能改善关节病理

Joyce Aarts, Arjan van Caam, Xinlai Chen, Renoud J Marijnissen, Monique M Helsen, Birgitte Walgreen, Elly L Vitters, Fons A van de Loo, Peter L van Lent, Peter M van der Kraan, Marije I Koenders

Targeting of fibroblast activation protein in rheumatoid arthritis patients: imaging and ex vivo photodynamic therapy

类风湿关节炎患者成纤维细胞活化蛋白的靶向治疗:成像和体外光动力疗法

Daphne N Dorst, Mark Rijpkema, Mijke Buitinga, Birgitte Walgreen, Monique M A Helsen, Evan Brennan, Christian Klein, Peter Laverman, Andreas Ramming, Christian Schmidkonz, Torsten Kuwert, Georg Schett, Peter M van der Kraan, Martin Gotthardt, Marije I Koenders

Systemic Resolvin E1 (RvE1) Treatment Does Not Ameliorate the Severity of Collagen-Induced Arthritis (CIA) in Mice: A Randomized, Prospective, and Controlled Proof of Concept Study

系统性 Resolvin E1 (RvE1) 治疗无法改善小鼠胶原诱导性关节炎 (CIA) 的严重程度:一项随机、前瞻性、对照的概念验证研究

Rafael Scaf de Molon, Rogier M Thurlings, Birgitte Walgreen, Monique M Helsen, Peter M van der Kraan, Joni Augusto Cirelli, Marije I Koenders

Fcγ receptor-mediated influx of S100A8/A9-producing neutrophils as inducer of bone erosion during antigen-induced arthritis

Fcγ 受体介导的 S100A8/A9 产生中性粒细胞的流入作为抗原诱发关节炎期间骨侵蚀的诱因

Irene Di Ceglie, Giuliana Ascone, Niels A J Cremers, Annet W Sloetjes, Birgitte Walgreen, Thomas Vogl, Johannes Roth, J Sjef Verbeek, Fons A J van de Loo, Marije I Koenders, Peter M van der Kraan, Arjen B Blom, Martijn H J van den Bosch, Peter L E M van Lent

Complementary action of granulocyte macrophage colony-stimulating factor and interleukin-17A induces interleukin-23, receptor activator of nuclear factor-κB ligand, and matrix metalloproteinases and drives bone and cartilage pathology in experimental arthritis: rationale for combination therapy in rheumatoid arthritis

粒细胞巨噬细胞集落刺激因子和白细胞介素-17A 的互补作用诱导白细胞介素-23、核因子-κB 配体的受体激活剂和基质金属蛋白酶,并驱动实验性关节炎中的骨和软骨病理:类风湿性关节炎联合治疗的原理

Annemarie E M van Nieuwenhuijze, Fons A van de Loo, Birgitte Walgreen, Miranda Bennink, Monique Helsen, Liduine van den Bersselaar, Ian P Wicks, Wim B van den Berg, Marije I Koenders

Interleukin-1 drives pathogenic Th17 cells during spontaneous arthritis in interleukin-1 receptor antagonist-deficient mice

在白细胞介素-1受体拮抗剂缺乏的小鼠中,白细胞介素-1在自发性关节炎期间驱动致病性Th17细胞

Marije I Koenders, Isabel Devesa, Renoud J Marijnissen, Shahla Abdollahi-Roodsaz, Annemieke M H Boots, Birgitte Walgreen, Franco E di Padova, Martin J H Nicklin, Leo A B Joosten, Wim B van den Berg